Page last updated: 2024-09-05

isospaglumic acid and tetrodotoxin

isospaglumic acid has been researched along with tetrodotoxin in 3 studies

Compound Research Comparison

Studies
(isospaglumic acid)
Trials
(isospaglumic acid)
Recent Studies (post-2010)
(isospaglumic acid)
Studies
(tetrodotoxin)
Trials
(tetrodotoxin)
Recent Studies (post-2010) (tetrodotoxin)
4693111612,992111,477

Protein Interaction Comparison

ProteinTaxonomyisospaglumic acid (IC50)tetrodotoxin (IC50)
Sodium channel protein type 9 subunit alphaHomo sapiens (human)0.0186

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19901 (33.33)18.7374
1990's1 (33.33)18.2507
2000's1 (33.33)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Artaud, F; Barbeito, L; Chéramy, A; Desce, JM; Galli, T; Glowsinki, J; Godeheu, G; Pittaluga, A1
Artaud, F; Barbeito, L; Chéramy, A; Glowinski, J; Godeheu, G; Pittaluga, A; Serval, V1
Bzdega, T; Cappiello, M; Neale, JH; Ramadan, E; Wroblewska, B; Zhao, J1

Other Studies

3 other study(ies) available for isospaglumic acid and tetrodotoxin

ArticleYear
Specific role of N-acetyl-aspartyl-glutamate in the in vivo regulation of dopamine release from dendrites and nerve terminals of nigrostriatal dopaminergic neurons in the cat.
    Neuroscience, 1991, Volume: 42, Issue:1

    Topics: Animals; Cats; Corpus Striatum; Dendrites; Dipeptides; Dopamine; Female; Ibotenic Acid; Male; N-Methylaspartate; Nerve Endings; Neurons; Substantia Nigra; Tetrodotoxin

1991
Depolarization-evoked release of N-acetyl-L-aspartyl-L-glutamate from rat brain synaptosomes.
    European journal of pharmacology, 1988, Dec-13, Volume: 158, Issue:3

    Topics: Animals; Brain; Calcimycin; Calcium; Diencephalon; Dipeptides; Male; Mesencephalon; Potassium; Rats; Rats, Inbred Strains; Synaptosomes; Tetrodotoxin; Veratridine

1988
NAAG inhibits KCl-induced [(3)H]-GABA release via mGluR3, cAMP, PKA and L-type calcium conductance.
    The European journal of neuroscience, 2001, Volume: 13, Issue:2

    Topics: Animals; Calcium Channel Blockers; Calcium Channels, L-Type; Cells, Cultured; Cerebral Cortex; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Dipeptides; gamma-Aminobutyric Acid; Neurons; Neuroprotective Agents; Nifedipine; Potassium Chloride; Presynaptic Terminals; Rats; Receptors, Metabotropic Glutamate; Synaptic Transmission; Tetrodotoxin; Tritium

2001