isoproterenol and 3-nitrotyrosine

isoproterenol has been researched along with 3-nitrotyrosine in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	2 (100.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Carretero, OA; Lin, C; Pagano, PJ; Rhaleb, NE; Sun, Y; Wang, F; Xu, J; Yang, JJ; Yang, XP	1
Gao, E; Grunwald, Z; Hu, A; Jiao, X; Koch, WJ; Ma, XL; Sharifi-Azad, S; Sun, JZ	1

Other Studies

2 other study(ies) available for isoproterenol and 3-nitrotyrosine

Article	Year
Lack of inducible NO synthase reduces oxidative stress and enhances cardiac response to isoproterenol in mice with deoxycorticosterone acetate-salt hypertension. Hypertension (Dallas, Tex. : 1979), 2005, Volume: 46, Issue:6 Topics: Aldehydes; Animals; Blood Pressure; Cardiotonic Agents; Desoxycorticosterone; Heart; Hypertension; Hypertrophy, Left Ventricular; Isoproterenol; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocardium; Nitric Oxide Synthase Type II; Nitric Oxide Synthase Type III; Oxidative Stress; Sodium Chloride; Tyrosine	2005
Chronic beta-adrenergic receptor stimulation induces cardiac apoptosis and aggravates myocardial ischemia/reperfusion injury by provoking inducible nitric-oxide synthase-mediated nitrative stress. The Journal of pharmacology and experimental therapeutics, 2006, Volume: 318, Issue:2 Topics: Adrenergic beta-Agonists; Animals; Apoptosis; Blotting, Western; C-Reactive Protein; Heart; Immunohistochemistry; In Situ Nick-End Labeling; Isoproterenol; Mice; Mice, Inbred C57BL; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Nitrates; Nitric Oxide; Nitric Oxide Synthase Type II; RNA; Stress, Physiological; Tyrosine	2006

Article

Year

Lack of inducible NO synthase reduces oxidative stress and enhances cardiac response to isoproterenol in mice with deoxycorticosterone acetate-salt hypertension.

Hypertension (Dallas, Tex. : 1979), 2005, Volume: 46, Issue:6

Topics: Aldehydes; Animals; Blood Pressure; Cardiotonic Agents; Desoxycorticosterone; Heart; Hypertension; Hypertrophy, Left Ventricular; Isoproterenol; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocardium; Nitric Oxide Synthase Type II; Nitric Oxide Synthase Type III; Oxidative Stress; Sodium Chloride; Tyrosine

2005

Chronic beta-adrenergic receptor stimulation induces cardiac apoptosis and aggravates myocardial ischemia/reperfusion injury by provoking inducible nitric-oxide synthase-mediated nitrative stress.

The Journal of pharmacology and experimental therapeutics, 2006, Volume: 318, Issue:2

Topics: Adrenergic beta-Agonists; Animals; Apoptosis; Blotting, Western; C-Reactive Protein; Heart; Immunohistochemistry; In Situ Nick-End Labeling; Isoproterenol; Mice; Mice, Inbred C57BL; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Nitrates; Nitric Oxide; Nitric Oxide Synthase Type II; RNA; Stress, Physiological; Tyrosine

2006