Page last updated: 2024-08-24

imiquimod and u 0126

imiquimod has been researched along with u 0126 in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	1 (50.00)	2.80

Authors

Authors	Studies
Dranchak, PK; Huang, R; Inglese, J; Lamy, L; Oliphant, E; Queme, B; Tao, D; Wang, Y; Xia, M	1
Ariffin, JK; Fairlie, DP; Hohenhaus, DM; Iyer, A; Lim, J; Seow, V; Suen, JY; Sweet, MJ	1

Other Studies

2 other study(ies) available for imiquimod and u 0126

Article	Year
In vivo quantitative high-throughput screening for drug discovery and comparative toxicology. Disease models & mechanisms, 2023, 03-01, Volume: 16, Issue:3 Topics: Animals; Caenorhabditis elegans; Drug Discovery; High-Throughput Screening Assays; Humans; Proteomics; Small Molecule Libraries	2023
Inflammatory responses induced by lipopolysaccharide are amplified in primary human monocytes but suppressed in macrophages by complement protein C5a. Journal of immunology (Baltimore, Md. : 1950), 2013, Oct-15, Volume: 191, Issue:8 Topics: Aminoquinolines; Butadienes; Cells, Cultured; Complement C5a; Extracellular Signal-Regulated MAP Kinases; Granulocyte-Macrophage Colony-Stimulating Factor; GTP-Binding Protein alpha Subunits, Gi-Go; Humans; Imiquimod; Inflammation; Interleukin-10; Interleukin-6; JNK Mitogen-Activated Protein Kinases; Lipopolysaccharides; Macrophage Colony-Stimulating Factor; Macrophages; MAP Kinase Signaling System; Mitogen-Activated Protein Kinase Kinases; Monocytes; Nitriles; Pertussis Toxin; Phosphatidylinositol 3-Kinases; Poly I-C; Proto-Oncogene Proteins c-raf; Receptor, Anaphylatoxin C5a; Receptors, Complement; Salmonella typhimurium; Toll-Like Receptor 4; Tumor Necrosis Factors	2013

Article

Year

In vivo quantitative high-throughput screening for drug discovery and comparative toxicology.

Disease models & mechanisms, 2023, 03-01, Volume: 16, Issue:3

Topics: Animals; Caenorhabditis elegans; Drug Discovery; High-Throughput Screening Assays; Humans; Proteomics; Small Molecule Libraries

2023

Inflammatory responses induced by lipopolysaccharide are amplified in primary human monocytes but suppressed in macrophages by complement protein C5a.

Journal of immunology (Baltimore, Md. : 1950), 2013, Oct-15, Volume: 191, Issue:8

Topics: Aminoquinolines; Butadienes; Cells, Cultured; Complement C5a; Extracellular Signal-Regulated MAP Kinases; Granulocyte-Macrophage Colony-Stimulating Factor; GTP-Binding Protein alpha Subunits, Gi-Go; Humans; Imiquimod; Inflammation; Interleukin-10; Interleukin-6; JNK Mitogen-Activated Protein Kinases; Lipopolysaccharides; Macrophage Colony-Stimulating Factor; Macrophages; MAP Kinase Signaling System; Mitogen-Activated Protein Kinase Kinases; Monocytes; Nitriles; Pertussis Toxin; Phosphatidylinositol 3-Kinases; Poly I-C; Proto-Oncogene Proteins c-raf; Receptor, Anaphylatoxin C5a; Receptors, Complement; Salmonella typhimurium; Toll-Like Receptor 4; Tumor Necrosis Factors

2013