ici 118551 and ovalbumin

ici 118551 has been researched along with ovalbumin in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (16.67)18.2507
2000's4 (66.67)29.6817
2010's1 (16.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bolton, PB; Lefevre, P; McDonald, DM1
Bond, RA; Dudekula, NB; Knoll, BJ; Lin, R; Nguyen, LP; Parra, S; Peng, H; Shardonofsky, F1
Maestroni, GJ; Manni, M1
Ammar-Aouchiche, Z; Bond, RA; Clement, C; Dickey, BF; Ehre, C; Frieske, JM; Ho, SB; Kesimer, M; Knoll, BJ; Nguyen, LP; Omoluabi, O; Parra, S; Tuvim, MJ1
Chupp, GL1
Kuda, Y; Kurata, Y; Shibamoto, T; Tanida, M; Yang, W; Zhang, T1

Other Studies

6 other study(ies) available for ici 118551 and ovalbumin

ArticleYear
Salmeterol reduces early- and late-phase plasma leakage and leukocyte adhesion in rat airways.
    American journal of respiratory and critical care medicine, 1997, Volume: 155, Issue:4

    Topics: Adrenergic beta-2 Receptor Antagonists; Adrenergic beta-Agonists; Adrenergic beta-Antagonists; Albuterol; Allergens; Animals; Capillary Permeability; Cell Adhesion; Leukocytes; Male; Ovalbumin; Premedication; Propanolamines; Rats; Rats, Inbred BN; Receptors, Adrenergic, beta-2; Respiratory Hypersensitivity; Salmeterol Xinafoate; Time Factors; Trachea

1997
Changes in beta 2-adrenoceptor and other signaling proteins produced by chronic administration of 'beta-blockers' in a murine asthma model.
    Pulmonary pharmacology & therapeutics, 2008, Volume: 21, Issue:1

    Topics: Adrenergic beta-Antagonists; Airway Resistance; Animals; Asthma; Blotting, Western; Bronchial Provocation Tests; Drug Administration Schedule; Fluorescent Antibody Technique; Intracellular Signaling Peptides and Proteins; Male; Methacholine Chloride; Metoprolol; Mice; Mice, Inbred BALB C; Nadolol; Ovalbumin; Propanolamines; Receptors, Adrenergic, beta-2

2008
Sympathetic nervous modulation of the skin innate and adaptive immune response to peptidoglycan but not lipopolysaccharide: involvement of beta-adrenoceptors and relevance in inflammatory diseases.
    Brain, behavior, and immunity, 2008, Volume: 22, Issue:1

    Topics: Adaptation, Physiological; Adrenergic beta-Antagonists; Animals; Betaxolol; Cell Movement; Dendritic Cells; Female; Immunity, Innate; Immunization; Immunologic Memory; Inflammation; Injections, Intradermal; Interferon-beta; Ligands; Lipopolysaccharides; Mice; Mice, Inbred BALB C; Mice, Transgenic; Ovalbumin; Peptidoglycan; Propanolamines; Propranolol; Receptors, Adrenergic, beta; Receptors, CXCR3; Skin; Skin Diseases; Sympathetic Nervous System; Th1 Cells

2008
Chronic exposure to beta-blockers attenuates inflammation and mucin content in a murine asthma model.
    American journal of respiratory cell and molecular biology, 2008, Volume: 38, Issue:3

    Topics: Administration, Oral; Adrenergic beta-Antagonists; Animals; Asthma; Disease Models, Animal; Female; Inflammation; Infusion Pumps; Injections, Intraperitoneal; Ligands; Male; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mucins; Nadolol; Ovalbumin; Propanolamines; Specific Pathogen-Free Organisms

2008
Say what, beta-blockers for asthma?
    American journal of respiratory cell and molecular biology, 2008, Volume: 38, Issue:3

    Topics: Administration, Oral; Adrenergic beta-Antagonists; Animals; Asthma; Bronchoalveolar Lavage Fluid; Cytokines; Disease Models, Animal; Infusion Pumps; Injections, Intraperitoneal; Mice; Mucins; Nadolol; Ovalbumin; Propanolamines; Reproducibility of Results

2008
Blockade of β2-adrenoceptor, rather than β1-adrenoceptor, deteriorates cardiac anaphylaxis in isolated blood-perfused rat hearts.
    Cardiology journal, 2017, Volume: 24, Issue:4

    Topics: Adrenergic beta-1 Receptor Antagonists; Adrenergic beta-2 Receptor Antagonists; Anaphylaxis; Animals; Atenolol; Coronary Vasospasm; Coronary Vessels; Disease Models, Animal; Isolated Heart Preparation; Male; Myocardial Contraction; Ovalbumin; Propanolamines; Rats, Wistar; Receptors, Adrenergic, beta-1; Receptors, Adrenergic, beta-2; Time Factors; Vasoconstriction; Ventricular Dysfunction, Left; Ventricular Function, Left; Ventricular Pressure

2017