hypochlorous acid has been researched along with aspirin in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (20.00) | 18.7374 |
1990's | 2 (40.00) | 18.2507 |
2000's | 1 (20.00) | 29.6817 |
2010's | 1 (20.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Husney, RM; Sagone, AL | 1 |
Berzhitskaia, VV; Murina, MA; Roshchupkin, DI; Sokolov, AIu | 1 |
Hammerschmidt, S; Wahn, H | 2 |
Bauer, G | 1 |
5 other study(ies) available for hypochlorous acid and aspirin
Article | Year |
---|---|
Oxidation of salicylates by stimulated granulocytes: evidence that these drugs act as free radical scavengers in biological systems.
Topics: Aspirin; Benzoates; Benzoic Acid; Chloramines; Free Radicals; Granulocytes; Hydrogen Peroxide; Hydroxides; Hypochlorous Acid; Oxidation-Reduction; Salicylates; Salicylic Acid; Superoxides; Thiourea; Xanthine Oxidase; Xanthines | 1987 |
[Changes in the initial platelet aggregation during storage of the platelet concentrate and exposure to the myeloperoxidase reaction products].
Topics: Animals; Aspirin; beta-Alanine; Blood Platelets; Blood Preservation; Calcium; Humans; Hypochlorous Acid; In Vitro Techniques; Nephelometry and Turbidimetry; Peroxidase; Platelet Aggregation; Platelet Aggregation Inhibitors; Rabbits; Thrombin | 1997 |
Inhibition of PMN- and HOC1-induced vascular injury in isolated rabbit lungs by acetylsalicylic acid: a possible link between neutrophil-derived oxidative stress and eicosanoid metabolism?
Topics: Animals; Aspirin; Capillaries; Eicosanoids; Female; Humans; Hypochlorous Acid; In Vitro Techniques; Lung; Male; Neutrophils; Oxidative Stress; Peroxidase; Pulmonary Circulation; Rabbits; Regression Analysis | 1998 |
Influence of cyclooxygenase and lipoxygenase inhibitors on oxidative stress-induced lung injury.
Topics: Animals; Antioxidants; Aspirin; Caffeic Acids; Capillary Permeability; Cyclooxygenase Inhibitors; Hypochlorous Acid; Lipoxygenase Inhibitors; Lung; Oxidative Stress; Pulmonary Wedge Pressure; Rabbits | 2001 |
Increasing the endogenous NO level causes catalase inactivation and reactivation of intercellular apoptosis signaling specifically in tumor cells.
Topics: Apoptosis; Aspirin; Catalase; Cell Line, Tumor; fas Receptor; Gene Expression Regulation, Neoplastic; Humans; Hydrogen Peroxide; Hypochlorous Acid; Metalloporphyrins; Neoplasms; Nitrates; Nitric Oxide; Peroxynitrous Acid; Proton Pumps; Signal Transduction; Singlet Oxygen | 2015 |