hydralazine has been researched along with angiotensin ii, des-phe(8)- in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Ferrario, CM; Gallagher, PE; Geary, RL; Igase, M; Strawn, WB | 1 |
| Al-Saleh, FM; Benter, IF; Chappell, MC; Diz, DI; Raghupathy, R; Yousif, MH | 1 |
| Alves, MN; Campagnole-Santos, MJ; de Almeida, PW; Dos Santos, RA; Gavioli, M; Greco, L; Guatimosim, S; Jesus, IC; Lima, Rde F; Melo, MB; Mitraud, L; Nocchi, E; Parreira, A; Resende, RR; Santiago, NM | 1 |
3 other study(ies) available for hydralazine and angiotensin ii, des-phe(8)-
| Article | Year |
|---|---|
Angiotensin II AT1 receptors regulate ACE2 and angiotensin-(1-7) expression in the aorta of spontaneously hypertensive rats.
Topics: Adrenergic beta-Antagonists; Angiotensin I; Angiotensin II; Angiotensin II Type 1 Receptor Blockers; Angiotensin-Converting Enzyme 2; Animals; Aorta, Thoracic; Atenolol; Carboxypeptidases; Carotid Arteries; Gene Expression Regulation, Enzymologic; Hydralazine; Hypertension; Imidazoles; Male; Olmesartan Medoxomil; Peptide Fragments; Peptidyl-Dipeptidase A; Rats; Rats, Inbred SHR; Receptor, Angiotensin, Type 1; RNA, Messenger; Tetrazoles; Vasodilator Agents | 2005 |
Angiotensin-(1-7) blockade attenuates captopril- or hydralazine-induced cardiovascular protection in spontaneously hypertensive rats treated with NG-nitro-L-arginine methyl ester.
Topics: Angiotensin I; Angiotensin II; Animals; Antihypertensive Agents; Blood Glucose; Blood Pressure; Brain; Captopril; Cytokines; Heart; Hydralazine; Hypertension; In Vitro Techniques; Insulin; Kidney; Leptin; Male; Myocardial Contraction; Myocardial Reperfusion Injury; NG-Nitroarginine Methyl Ester; Peptide Fragments; Perfusion; Proteinuria; Rats; Rats, Inbred SHR; Ventricular Function, Left | 2011 |
Beneficial effects of angiotensin-(1-7) against deoxycorticosterone acetate-induced diastolic dysfunction occur independently of changes in blood pressure.
Topics: Angiotensin I; Animals; Blood Pressure; Calcium; Calcium Signaling; Desoxycorticosterone Acetate; Disease Models, Animal; Dose-Response Relationship, Drug; Heart Failure, Diastolic; Hydralazine; Hypertension; Male; Peptide Fragments; Rats; Rats, Sprague-Dawley; Rats, Transgenic | 2015 |