Compounds > hyaluronoglucosaminidase
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hyaluronoglucosaminidase and staurosporine

hyaluronoglucosaminidase has been researched along with staurosporine in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (20.00)18.2507
2000's4 (80.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Carey, G; Chang, NS; Chu, E; Ou, M; Pratt, N1
Chang, NS3
Barak, O; Bhatnagar, RS; Csóka, AB; Nicoll, SB; Stern, R1

Other Studies

5 other study(ies) available for hyaluronoglucosaminidase and staurosporine

ArticleYear
p53 overexpression and downregulation of inter-alpha-inhibitor are associated with hyaluronidase enhancement of TNF cytotoxicity in L929 fibroblasts.
    Cancer letters, 1998, Sep-11, Volume: 131, Issue:1

    Topics: alpha-Fetoproteins; Alpha-Globulins; Animals; Benzoquinones; Cell Survival; Down-Regulation; Enzyme Inhibitors; Fibroblasts; Genes, p53; Hyaluronoglucosaminidase; Lactams, Macrocyclic; Mice; Neoplasm Proteins; Quinones; Staurosporine; Trypsin Inhibitors; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha

1998
Hyaluronidase activation of c-Jun N-terminal kinase is necessary for protection of L929 fibrosarcoma cells from staurosporine-mediated cell death.
    Biochemical and biophysical research communications, 2001, May-04, Volume: 283, Issue:2

    Topics: Animals; Antineoplastic Agents; Cell Death; Cell Line; COS Cells; Drug Resistance; Enzyme Activation; Humans; Hyaluronoglucosaminidase; JNK Mitogen-Activated Protein Kinases; Male; Mice; Mitogen-Activated Protein Kinases; Staurosporine; Transforming Growth Factor beta; Tumor Cells, Cultured

2001
The non-ankyrin C terminus of Ikappa Balpha physically interacts with p53 in vivo and dissociates in response to apoptotic stress, hypoxia, DNA damage, and transforming growth factor-beta 1-mediated growth suppression.
    The Journal of biological chemistry, 2002, Mar-22, Volume: 277, Issue:12

    Topics: Animals; Ankyrins; Apoptosis; Cell Division; Cell Line; Cell Nucleus; COS Cells; Cytosol; DNA Damage; DNA-Binding Proteins; Dose-Response Relationship, Drug; Down-Regulation; Etoposide; Genes, Dominant; Humans; Hyaluronoglucosaminidase; Hypoxia; I-kappa B Proteins; Liver; Mice; Microscopy, Fluorescence; Mutagenesis, Site-Directed; NF-KappaB Inhibitor alpha; Phosphorylation; Precipitin Tests; Proline; Protein Binding; Protein Structure, Tertiary; Rats; Recombinant Proteins; Serine; Spleen; Staurosporine; Threonine; Time Factors; Transfection; Transforming Growth Factor beta; Transforming Growth Factor beta1; Tumor Cells, Cultured; Tumor Suppressor Protein p53; Ultraviolet Rays

2002
Hyaluronidases and CD44 undergo differential modulation during chondrogenesis.
    Biochemical and biophysical research communications, 2002, Apr-12, Volume: 292, Issue:4

    Topics: Cell Adhesion Molecules; Cell Differentiation; Cells, Cultured; Chondrogenesis; Culture Media, Serum-Free; Enzyme Inhibitors; Fibroblasts; Gene Expression Regulation; Humans; Hyaluronan Receptors; Hyaluronoglucosaminidase; Isoenzymes; Lactic Acid; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Staurosporine; Up-Regulation

2002
Transforming growth factor-beta1 blocks the enhancement of tumor necrosis factor cytotoxicity by hyaluronidase Hyal-2 in L929 fibroblasts.
    BMC cell biology, 2002, Apr-03, Volume: 3

    Topics: Animals; Antineoplastic Agents; Apoptosis; Base Sequence; Cell Adhesion Molecules; Cell Division; Cell Line; COS Cells; Fibroblasts; GPI-Linked Proteins; Hyaluronoglucosaminidase; Mice; Mitochondria; Molecular Sequence Data; NF-kappa B; Oxidoreductases; Protein Transport; Staurosporine; Transforming Growth Factor beta; Transforming Growth Factor beta1; Tumor Necrosis Factor-alpha; WW Domain-Containing Oxidoreductase

2002