ha 1004 has been researched along with 8-bromo cyclic adenosine monophosphate in 7 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (14.29) | 18.7374 |
1990's | 5 (71.43) | 18.2507 |
2000's | 1 (14.29) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Amador, M; Dani, JA; Moorman, JR; Pinkham, J; Reuhl, TO | 1 |
Berg, KL; Foster, DA; Joseph, C; Qureshi, SA; Spangler, R | 1 |
Arola, J; Heikkilä, P; Kahri, AI; Voutilainen, R | 1 |
Gubitz, AK; Kirkpatrick, KA; Kurokawa, M; Richardson, PJ; Widdowson, L | 1 |
Dohi, T; Minami, N; Miyasako, T; Morita, K; Suemitsu, T | 1 |
Jett, M; Yan, Z; Yang, DC | 1 |
Fukui, H; Horio, S; Kawakami, N; Miyoshi, K | 1 |
7 other study(ies) available for ha 1004 and 8-bromo cyclic adenosine monophosphate
Article | Year |
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Nicotinic acetylcholine receptors are directly affected by agents used to study protein phosphorylation.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 1-Methyl-3-isobutylxanthine; 8-Bromo Cyclic Adenosine Monophosphate; Animals; Cells, Cultured; Ion Channels; Isoquinolines; Membrane Potentials; Nicotinic Antagonists; Oocytes; Phosphorylation; Piperazines; Protein Kinase Inhibitors; Protein Kinases; Proteins; Receptors, Nicotinic; RNA, Messenger; Sulfonamides; Xenopus | 1992 |
Evidence that v-src and v-fps gene products use a protein kinase C-mediated pathway to induce expression of a transformation-related gene.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Animals; Bucladesine; Calcium Channel Blockers; Cell Transformation, Neoplastic; Chick Embryo; Gene Products, gag; Isoquinolines; Kinetics; Oncogene Protein pp60(v-src); Oncogenes; Phorbol Esters; Phosphorylation; Piperazines; Protein Kinase C; Protein Kinase Inhibitors; Protein Kinases; Retroviridae Proteins; RNA, Messenger; Signal Transduction; Sulfonamides; Transcription, Genetic | 1989 |
Protein kinase C signal transduction pathway in ACTH-induced growth effect of rat adrenocortical cells in primary culture.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Adrenal Cortex; Adrenocorticotropic Hormone; Alkaloids; Animals; Cell Division; Cells, Cultured; Corticosterone; Isoquinolines; Piperazines; Protein Kinase C; Rats; Rats, Sprague-Dawley; Signal Transduction; Staurosporine; Stimulation, Chemical; Sulfonamides; Tetradecanoylphorbol Acetate; Time Factors | 1994 |
Dual signalling by the adenosine A2a receptor involves activation of both N- and P-type calcium channels by different G proteins and protein kinases in the same striatal nerve terminals.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Acetylcholine; Adenosine; Agatoxins; Alkaloids; Animals; Antihypertensive Agents; Benzophenanthridines; Calcium Channel Blockers; Calcium Channels; Cholera Toxin; Cholinergic Fibers; Colforsin; Cyclic AMP-Dependent Protein Kinases; Enzyme Inhibitors; GTP-Binding Proteins; Isoquinolines; Neostriatum; Neurons; Phenanthridines; Phenethylamines; Piperazines; Potassium; Presynaptic Terminals; Protein Kinase C; Protein Kinases; Rats; Rats, Wistar; Receptors, Purinergic P1; Signal Transduction; Spider Venoms; Stereoisomerism; Sulfonamides; Tritium; Xanthines | 1996 |
Cyclic AMP enhances acetylcholine (ACh)-induced ion fluxes and catecholamine release by inhibiting Na+, K(+)-ATPase and participates in the responses to ACh in cultured bovine adrenal medullary chromaffin cells.
Topics: 8-Bromo Cyclic Adenosine Monophosphate; Acetylcholine; Adrenal Medulla; Animals; Calcium; Catecholamines; Cattle; Cells, Cultured; Colforsin; Cyclic AMP; Enzyme Inhibitors; Isoquinolines; Membrane Potentials; Ouabain; Protein Kinase Inhibitors; Receptors, Cholinergic; Sodium; Sodium-Potassium-Exchanging ATPase; Sulfonamides; Tetrodotoxin; Thionucleotides | 1995 |
Cholera toxin induces tumor necrosis factor alpha production in human monocytes.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Adenosine Diphosphate; Benzamides; Cells, Cultured; Cholera Toxin; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Dose-Response Relationship, Drug; Enzyme Inhibitors; Humans; Isoquinolines; Lymphocytes; Monocytes; Niacinamide; Poly(ADP-ribose) Polymerase Inhibitors; Protein Kinase C; Signal Transduction; Sphingosine; Sulfonamides; Transcription, Genetic; Tumor Necrosis Factor-alpha; Type C Phospholipases | 1999 |
Inhibition of histamine H1 receptor downregulation by KT5823, a protein kinase G inhibitor.
Topics: 8-Bromo Cyclic Adenosine Monophosphate; Animals; Carbazoles; CHO Cells; Cricetinae; Cyclic AMP-Dependent Protein Kinases; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Down-Regulation; Enzyme Activators; Histamine; Indoles; Isoquinolines; Protein Kinase C; Receptors, Histamine H1; Sulfonamides; Thionucleotides; Time Factors | 2003 |