ha 1004 and 8-bromo cyclic adenosine monophosphate

ha 1004 has been researched along with 8-bromo cyclic adenosine monophosphate in 7 studies

Research

Studies (7)

TimeframeStudies, this research(%)All Research%
pre-19901 (14.29)18.7374
1990's5 (71.43)18.2507
2000's1 (14.29)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Amador, M; Dani, JA; Moorman, JR; Pinkham, J; Reuhl, TO1
Berg, KL; Foster, DA; Joseph, C; Qureshi, SA; Spangler, R1
Arola, J; Heikkilä, P; Kahri, AI; Voutilainen, R1
Gubitz, AK; Kirkpatrick, KA; Kurokawa, M; Richardson, PJ; Widdowson, L1
Dohi, T; Minami, N; Miyasako, T; Morita, K; Suemitsu, T1
Jett, M; Yan, Z; Yang, DC1
Fukui, H; Horio, S; Kawakami, N; Miyoshi, K1

Other Studies

7 other study(ies) available for ha 1004 and 8-bromo cyclic adenosine monophosphate

ArticleYear
Nicotinic acetylcholine receptors are directly affected by agents used to study protein phosphorylation.
    Journal of neurophysiology, 1992, Volume: 68, Issue:2

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 1-Methyl-3-isobutylxanthine; 8-Bromo Cyclic Adenosine Monophosphate; Animals; Cells, Cultured; Ion Channels; Isoquinolines; Membrane Potentials; Nicotinic Antagonists; Oocytes; Phosphorylation; Piperazines; Protein Kinase Inhibitors; Protein Kinases; Proteins; Receptors, Nicotinic; RNA, Messenger; Sulfonamides; Xenopus

1992
Evidence that v-src and v-fps gene products use a protein kinase C-mediated pathway to induce expression of a transformation-related gene.
    Proceedings of the National Academy of Sciences of the United States of America, 1989, Volume: 86, Issue:18

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Animals; Bucladesine; Calcium Channel Blockers; Cell Transformation, Neoplastic; Chick Embryo; Gene Products, gag; Isoquinolines; Kinetics; Oncogene Protein pp60(v-src); Oncogenes; Phorbol Esters; Phosphorylation; Piperazines; Protein Kinase C; Protein Kinase Inhibitors; Protein Kinases; Retroviridae Proteins; RNA, Messenger; Signal Transduction; Sulfonamides; Transcription, Genetic

1989
Protein kinase C signal transduction pathway in ACTH-induced growth effect of rat adrenocortical cells in primary culture.
    The Journal of endocrinology, 1994, Volume: 141, Issue:2

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Adrenal Cortex; Adrenocorticotropic Hormone; Alkaloids; Animals; Cell Division; Cells, Cultured; Corticosterone; Isoquinolines; Piperazines; Protein Kinase C; Rats; Rats, Sprague-Dawley; Signal Transduction; Staurosporine; Stimulation, Chemical; Sulfonamides; Tetradecanoylphorbol Acetate; Time Factors

1994
Dual signalling by the adenosine A2a receptor involves activation of both N- and P-type calcium channels by different G proteins and protein kinases in the same striatal nerve terminals.
    Journal of neurochemistry, 1996, Volume: 67, Issue:1

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Acetylcholine; Adenosine; Agatoxins; Alkaloids; Animals; Antihypertensive Agents; Benzophenanthridines; Calcium Channel Blockers; Calcium Channels; Cholera Toxin; Cholinergic Fibers; Colforsin; Cyclic AMP-Dependent Protein Kinases; Enzyme Inhibitors; GTP-Binding Proteins; Isoquinolines; Neostriatum; Neurons; Phenanthridines; Phenethylamines; Piperazines; Potassium; Presynaptic Terminals; Protein Kinase C; Protein Kinases; Rats; Rats, Wistar; Receptors, Purinergic P1; Signal Transduction; Spider Venoms; Stereoisomerism; Sulfonamides; Tritium; Xanthines

1996
Cyclic AMP enhances acetylcholine (ACh)-induced ion fluxes and catecholamine release by inhibiting Na+, K(+)-ATPase and participates in the responses to ACh in cultured bovine adrenal medullary chromaffin cells.
    Journal of neural transmission. General section, 1995, Volume: 100, Issue:1

    Topics: 8-Bromo Cyclic Adenosine Monophosphate; Acetylcholine; Adrenal Medulla; Animals; Calcium; Catecholamines; Cattle; Cells, Cultured; Colforsin; Cyclic AMP; Enzyme Inhibitors; Isoquinolines; Membrane Potentials; Ouabain; Protein Kinase Inhibitors; Receptors, Cholinergic; Sodium; Sodium-Potassium-Exchanging ATPase; Sulfonamides; Tetrodotoxin; Thionucleotides

1995
Cholera toxin induces tumor necrosis factor alpha production in human monocytes.
    Molecular cell biology research communications : MCBRC, 1999, Volume: 2, Issue:2

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Adenosine Diphosphate; Benzamides; Cells, Cultured; Cholera Toxin; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Dose-Response Relationship, Drug; Enzyme Inhibitors; Humans; Isoquinolines; Lymphocytes; Monocytes; Niacinamide; Poly(ADP-ribose) Polymerase Inhibitors; Protein Kinase C; Signal Transduction; Sphingosine; Sulfonamides; Transcription, Genetic; Tumor Necrosis Factor-alpha; Type C Phospholipases

1999
Inhibition of histamine H1 receptor downregulation by KT5823, a protein kinase G inhibitor.
    Methods and findings in experimental and clinical pharmacology, 2003, Volume: 25, Issue:5

    Topics: 8-Bromo Cyclic Adenosine Monophosphate; Animals; Carbazoles; CHO Cells; Cricetinae; Cyclic AMP-Dependent Protein Kinases; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Down-Regulation; Enzyme Activators; Histamine; Indoles; Isoquinolines; Protein Kinase C; Receptors, Histamine H1; Sulfonamides; Thionucleotides; Time Factors

2003