Page last updated: 2024-09-03

glycyl-arginyl-glycyl-aspartyl-seryl-proline and am 281

glycyl-arginyl-glycyl-aspartyl-seryl-proline has been researched along with am 281 in 1 studies

Compound Research Comparison

Studies
(glycyl-arginyl-glycyl-aspartyl-seryl-proline)
Trials
(glycyl-arginyl-glycyl-aspartyl-seryl-proline)
Recent Studies (post-2010)
(glycyl-arginyl-glycyl-aspartyl-seryl-proline)
Studies
(am 281)
Trials
(am 281)
Recent Studies (post-2010) (am 281)
162120104039

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bahr, BA; Brown, QB; Karanian, DA; Makriyannis, A1

Other Studies

1 other study(ies) available for glycyl-arginyl-glycyl-aspartyl-seryl-proline and am 281

ArticleYear
Blocking cannabinoid activation of FAK and ERK1/2 compromises synaptic integrity in hippocampus.
    European journal of pharmacology, 2005, Jan-31, Volume: 508, Issue:1-3

    Topics: Animals; Arachidonic Acids; Cannabinoids; Dose-Response Relationship, Drug; Enzyme Activation; Female; Focal Adhesion Kinase 1; Focal Adhesion Protein-Tyrosine Kinases; Hippocampus; Immunoblotting; Male; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Morpholines; Oligopeptides; Organ Culture Techniques; Protein Kinases; Protein-Tyrosine Kinases; Pyrazoles; Rats; Rats, Sprague-Dawley; Receptor, Cannabinoid, CB1; Signal Transduction; Synapses; Time Factors

2005