glycerylphosphorylcholine has been researched along with gamma-aminobutyric acid in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (50.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Beani, L; Bianchi, C; Ferraro, L; Marani, L; Siniscalchi, A; Tanganelli, S | 1 |
Hayakawa, T; Kohmura, E; Nonaka, M; Wakayama, A; Yamashita, T; Yoshimine, T | 1 |
Galloway, MP; Ghoddoussi, F; Hall, CS; Perrine, SA; Sajja, VS; VandeVord, PJ | 1 |
Brodsky, JL; Chiang, A; Chung, WJ; Denny, RA; Goeckeler-Fried, JL; Havasi, V; Hong, JS; Keeton, AB; Mazur, M; Piazza, GA; Plyler, ZE; Rasmussen, L; Rowe, SM; Sorscher, EJ; Weissman, AM; White, EL | 1 |
4 other study(ies) available for glycerylphosphorylcholine and gamma-aminobutyric acid
Article | Year |
---|---|
Evidence for an in vivo and in vitro modulation of endogenous cortical GABA release by alpha-glycerylphosphorylcholine.
Topics: Animals; Atropine; Cerebral Cortex; Drug Synergism; gamma-Aminobutyric Acid; Glycerylphosphorylcholine; In Vitro Techniques; Injections, Intraperitoneal; Kinetics; Male; Mecamylamine; Prazosin; Rats; Rats, Sprague-Dawley; Tetrodotoxin | 1996 |
Changes in brain organic osmolytes in experimental cerebral ischemia.
Topics: Alanine; Amino Acids; Animals; Aspartic Acid; Brain Chemistry; Brain Ischemia; Creatine; Disease Models, Animal; gamma-Aminobutyric Acid; Glutamic Acid; Glutamine; Glycerylphosphorylcholine; Magnetic Resonance Spectroscopy; Male; Methylamines; Phosphocreatine; Rats; Rats, Sprague-Dawley; Taurine | 1998 |
Blast neurotrauma impairs working memory and disrupts prefrontal myo-inositol levels in rats.
Topics: Animals; Betaine; Blast Injuries; Brain Injuries; Creatine; Ethanolamines; gamma-Aminobutyric Acid; Glutamic Acid; Glycerylphosphorylcholine; Inositol; Lactic Acid; Male; Memory, Short-Term; Pattern Recognition, Physiological; Prefrontal Cortex; Rats; Rats, Sprague-Dawley | 2014 |
Increasing the Endoplasmic Reticulum Pool of the F508del Allele of the Cystic Fibrosis Transmembrane Conductance Regulator Leads to Greater Folding Correction by Small Molecule Therapeutics.
Topics: Alleles; Benzoates; Cells, Cultured; Cystic Fibrosis; Cystic Fibrosis Transmembrane Conductance Regulator; Endoplasmic Reticulum; Furans; Gene Deletion; HEK293 Cells; HeLa Cells; High-Throughput Screening Assays; Humans; Hydroxamic Acids; Microscopy, Fluorescence; Protein Folding; Protein Structure, Tertiary; Pyrazoles; RNA, Messenger; Small Molecule Libraries; Ubiquitination; Vorinostat | 2016 |