glutamic acid has been researched along with brevetoxin in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (33.33) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Berman, FW; Murray, TF | 2 |
| Baden, DG; Dravid, SM; Murray, TF | 1 |
3 other study(ies) available for glutamic acid and brevetoxin
| Article | Year |
|---|---|
Brevetoxins cause acute excitotoxicity in primary cultures of rat cerebellar granule neurons.
Topics: Animals; Aspartic Acid; Cell Death; Cell Survival; Cells, Cultured; Cerebellum; Glutamic Acid; L-Lactate Dehydrogenase; Marine Toxins; Neurons; Neurotoxins; Oxocins; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate | 1999 |
Brevetoxin-induced autocrine excitotoxicity is associated with manifold routes of Ca2+ influx.
Topics: 2-Amino-5-phosphonovalerate; Aniline Compounds; Animals; Autocrine Communication; Biological Transport; Calcium; Calcium Channel Blockers; Calcium Channels, L-Type; Cells, Cultured; Cerebellum; Dextrorphan; Dizocilpine Maleate; Dose-Response Relationship, Drug; Excitatory Amino Acid Antagonists; Fluorescent Dyes; Glutamic Acid; Marine Toxins; Neurons; Neurotoxins; Nifedipine; Oxocins; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; Sodium Channels; Thiourea; Xanthenes | 2000 |
Brevetoxin activation of voltage-gated sodium channels regulates Ca dynamics and ERK1/2 phosphorylation in murine neocortical neurons.
Topics: Animals; Brain-Derived Neurotrophic Factor; Calcium; Calcium Signaling; Cells, Cultured; Cyclic AMP Response Element-Binding Protein; Enzyme Inhibitors; Enzymes; Excitatory Amino Acid Antagonists; Glutamic Acid; Ion Channel Gating; Marine Toxins; Mice; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Neocortex; Neurons; Neurotoxins; Oxocins; Phosphorylation; Sodium Channels | 2004 |