glucose, (beta-d)-isomer has been researched along with imiquimod in 12 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 10 (83.33) | 24.3611 |
| 2020's | 2 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Chan, ZW; Chui, LG; Feingold, KR; Grunfeld, C; Kazemi, MR; Londos, C; Magra, AL; McDonald, CM; Patzek, SM; Shigenaga, JK | 1 |
| Chandra, N; Cote, LC; Doncel, GF; Joseph, T; Schriver, SD; Yousefieh, N; Zalenskaya, IA | 1 |
| Cross, AS; Feingold, KR; Grunfeld, C; Moser, A; Shigenaga, JK | 1 |
| Huo, R; Li, H; Li, N; Shen, B; Sun, Y; Wu, P; Zhai, T; Zhang, J; Zhou, Z; Zhu, X | 1 |
| Katayama, H; Koyama, M; Nakano, A; Nohmi, K; Saito, M; Sakashita, Y; Shintaku, H; Tachibana, D; Terada, H; Tokuhara, D | 1 |
| Di, T; Li, P; Liang, D; Liu, X; Wang, Y; Zhao, J | 1 |
| Cao, N; Chen, T; Fu, LX; Lu, YH; Yin, B; Zhang, LW; Zhou, PM | 1 |
| Albanesi, C; Avigliano, L; Cavani, A; Madonna, S; Mauriello, A; Melino, G; Palombo, R; Savini, I; Terrinoni, A | 1 |
| Fan, S; Li, Y; Liu, W; Wang, N; Yang, X; Zheng, X; Zhu, Y | 1 |
| Chen, Y; Fang, W; He, S; Huang, L; Leng, Y; Li, B; Li, Y; Liu, D; Liu, G; Liu, R; Liu, X; Wang, R; Yang, Z; Ye, C; Yin, H | 1 |
| Bai, X; Dang, E; Luo, Y; Wang, G; Yang, L; Yu, C; Zhi, D | 1 |
| Fu, J; Li, P; Wang, Y; Zeng, Z; Zhang, L | 1 |
12 other study(ies) available for glucose, (beta-d)-isomer and imiquimod
| Article | Year |
|---|---|
ADRP/ADFP and Mal1 expression are increased in macrophages treated with TLR agonists.
Topics: Aminoquinolines; Animals; Atherosclerosis; Cholesterol Esters; Fatty Acid-Binding Proteins; Humans; Imiquimod; Lipopolysaccharides; Macrophage Activation; Macrophages; Membrane Proteins; Mice; Neoplasm Proteins; Perilipin-2; Poly I-C; Toll-Like Receptors; Triglycerides; Zymosan | 2010 |
Induction of cyclooxygenase (COX)-2 in human vaginal epithelial cells in response to TLR ligands and TNF-α.
Topics: Aminoquinolines; Cell Line; Cyclooxygenase 2; Dinoprostone; Epithelial Cells; Female; Humans; Imiquimod; Ligands; Lipopeptides; Lipopolysaccharides; Polydeoxyribonucleotides; RNA, Messenger; Teichoic Acids; Toll-Like Receptors; Tumor Necrosis Factor-alpha; Up-Regulation; Vagina; Zymosan | 2012 |
Angiopoietin like protein 4 expression is decreased in activated macrophages.
Topics: Aminoquinolines; Angiopoietin-Like Protein 4; Angiopoietins; Animals; Female; Imiquimod; Lipopolysaccharides; Macrophage Activation; Macrophages, Peritoneal; Mice; Mice, Inbred C57BL; Poly I-C; Toll-Like Receptors; Zymosan | 2012 |
Paeoniflorin inhibits skin lesions in imiquimod-induced psoriasis-like mice by downregulating inflammation.
Topics: Aminoquinolines; Animals; Anti-Inflammatory Agents; Cytokines; Female; Glucosides; Imiquimod; Macrophages; Mice, Inbred BALB C; Monoterpenes; Neutrophils; Psoriasis; Skin | 2015 |
Zymosan Induces Immune Responses Comparable with Those of Adults in Monocytes, Dendritic Cells, and Monocyte-Derived Dendritic Cells from Cord Blood.
Topics: Adjuvants, Immunologic; Adult; Aminoquinolines; Cells, Cultured; Cytokines; Dendritic Cells; Fetal Blood; Flagellin; Flow Cytometry; Humans; Imiquimod; Infant, Newborn; Lipopeptides; Lipopolysaccharides; Monocytes; Oligodeoxyribonucleotides; Poly I-C; Zymosan | 2015 |
Paeoniflorin inhibits imiquimod-induced psoriasis in mice by regulating Th17 cell response and cytokine secretion.
Topics: Aminoquinolines; Animals; Cell Differentiation; Cell Proliferation; Cell Survival; Cytokines; Gene Expression Regulation; Glucosides; Imiquimod; Keratinocytes; Mice; Monoterpenes; Nuclear Receptor Subfamily 1, Group F, Member 3; Phosphorylation; Psoriasis; RNA, Messenger; Skin; STAT3 Transcription Factor; Th17 Cells | 2016 |
Paeoniflorin suppresses inflammatory response in imiquimod-induced psoriasis-like mice and peripheral blood mononuclear cells (PBMCs) from psoriasis patients.
Topics: Aminoquinolines; Animals; Anti-Inflammatory Agents, Non-Steroidal; Cells, Cultured; Glucosides; Humans; Imiquimod; Inflammation Mediators; Leukocytes, Mononuclear; Male; Mice; Mice, Inbred BALB C; Monoterpenes; Psoriasis; Random Allocation | 2016 |
Luteolin-7-glucoside inhibits IL-22/STAT3 pathway, reducing proliferation, acanthosis, and inflammation in keratinocytes and in mouse psoriatic model.
Topics: Acanthosis Nigricans; Aminoquinolines; Animals; Cell Differentiation; Cell Nucleus; Cell Proliferation; Cells, Cultured; Cellular Senescence; Disease Models, Animal; Glucosides; Humans; Imiquimod; Immunohistochemistry; Inflammation; Interleukin-22; Interleukins; Keratinocytes; Lipids; Luteolin; Mice, Inbred C57BL; Oxidation-Reduction; Phenotype; Protein Transport; Psoriasis; Signal Transduction; STAT3 Transcription Factor | 2016 |
Multiple Immunosuppressive Effects of CpG-c41 on Intracellular TLR-Mediated Inflammation.
Topics: Aminoquinolines; Animals; Blotting, Western; Enzyme-Linked Immunosorbent Assay; Female; Fluorescent Antibody Technique; Imidazoles; Imiquimod; Immunity, Innate; Immunosuppressive Agents; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Oligodeoxyribonucleotides; RAW 264.7 Cells; Signal Transduction; Toll-Like Receptor 3; Toll-Like Receptor 7; Toll-Like Receptor 8; Toll-Like Receptor 9; Zymosan | 2017 |
Total glucosides of paeony attenuates animal psoriasis induced inflammatory response through inhibiting STAT1 and STAT3 phosphorylation.
Topics: Animals; Cytokines; Disease Models, Animal; Female; Glucosides; Guinea Pigs; Imiquimod; Male; Mice, Inbred BALB C; Nuclear Receptor Subfamily 1, Group F, Member 3; Paeonia; Phosphorylation; Plant Roots; Psoriasis; STAT1 Transcription Factor; STAT3 Transcription Factor | 2019 |
Anti-psoriatic properties of paeoniflorin: suppression of the NF-kappaB pathway and Keratin 17.
Topics: Administration, Cutaneous; Animals; Anti-Inflammatory Agents, Non-Steroidal; Cell Proliferation; Cytokines; Down-Regulation; Female; Glucosides; HaCaT Cells; Humans; Imiquimod; Keratin-17; Keratinocytes; Keratins; Mice; Monoterpenes; NF-kappa B; Psoriasis; Signal Transduction | 2020 |
4'-O-β-D-glucosyl-5-O-methylvisamminol ameliorates imiquimod-induced psoriasis-like dermatitis and inhibits inflammatory cytokines production by suppressing the NF-κB and MAPK signaling pathways.
Topics: Animals; Chromones; Cytokines; Dermatitis; Glucosides; Imiquimod; Inflammation; Lipopolysaccharides; MAP Kinase Signaling System; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis | 2020 |