glucosamine has been researched along with thapsigargin in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Champattanachai, V; Chatham, JC; Marchase, RB; Nagy, T | 1 |
Chen, Q; Gao, XY; Kang, YM; Li, YH; Shi, CX; Shu, XD; Wang, JJ; Xiong, XQ; Zhao, MX; Zhu, GQ | 1 |
Beguinot, F; D'Esposito, V; Di Jeso, B; Fiory, F; Formisano, P; Longo, M; Miele, C; Nigro, C; Raciti, GA; Spinelli, R; Zatterale, F | 1 |
Beriault, DR; Dang, VT; McAlpine, CS; Petlura, CI; Shi, Y; Werstuck, GH; Zhong, LH | 1 |
4 other study(ies) available for glucosamine and thapsigargin
Article | Year |
---|---|
Glucosamine inhibits angiotensin II-induced cytoplasmic Ca2+ elevation in neonatal cardiomyocytes via protein-associated O-linked N-acetylglucosamine.
Topics: Acetylglucosamine; Angiotensin II; Animals; Animals, Newborn; Calcium; Calcium Signaling; Cells, Cultured; Cytoplasm; Down-Regulation; Drug Interactions; Enzyme Inhibitors; Glucosamine; Hexosamines; Homeostasis; Myocytes, Cardiac; Rats; Rats, Sprague-Dawley; Thapsigargin; Up-Regulation; Vasoconstrictor Agents | 2006 |
β-aminoisobutyric acid attenuates hepatic endoplasmic reticulum stress and glucose/lipid metabolic disturbance in mice with type 2 diabetes.
Topics: Administration, Oral; Aminoisobutyric Acids; AMP-Activated Protein Kinases; Animals; Apoptosis; Blood Glucose; Blotting, Western; Carbohydrate Metabolism; Cholesterol; Diabetes Mellitus, Experimental; Diet, High-Fat; Endoplasmic Reticulum Stress; Glucosamine; Hep G2 Cells; Humans; Immunohistochemistry; Insulin Resistance; Lipid Metabolism; Liver; Mice; Phosphorylation; Real-Time Polymerase Chain Reaction; RNA Interference; RNA, Small Interfering; Signal Transduction; Thapsigargin; Triglycerides; Tunicamycin | 2016 |
Pathologic endoplasmic reticulum stress induced by glucotoxic insults inhibits adipocyte differentiation and induces an inflammatory phenotype.
Topics: 3T3-L1 Cells; Adipocytes; Adult; Animals; Blotting, Western; Cell Differentiation; Cell Line; Cells, Cultured; Cytokines; Endoplasmic Reticulum Stress; Gene Expression; Glucosamine; Humans; Inflammation Mediators; Mice; Middle Aged; NF-kappa B; Phenotype; Phenylbutyrates; Phenylenediamines; Reverse Transcriptase Polymerase Chain Reaction; Thapsigargin; Unfolded Protein Response | 2016 |
Glucosamine induces ER stress by disrupting lipid-linked oligosaccharide biosynthesis and N-linked protein glycosylation.
Topics: Animals; Cell Line; Dithiothreitol; Endoplasmic Reticulum Stress; Enzyme Inhibitors; Fibroblasts; Glucosamine; Glycosylation; Lipopolysaccharides; Mice; Phenylbutyrates; Thapsigargin; Unfolded Protein Response | 2017 |