gastrin-releasing-peptide and camostat

gastrin-releasing-peptide has been researched along with camostat* in 2 studies

Other Studies

2 other study(ies) available for gastrin-releasing-peptide and camostat

Article	Year
Gastrin-releasing peptide and CCK after intraduodenal inhibition of proteases in dogs. International journal of pancreatology : official journal of the International Association of Pancreatology, 1994, Volume: 15, Issue:3 The intraduodenal application of a potent protease inhibitor (camostate, 600 mg) causes a significant increase in basal pancreatic secretion in the manner of a negative-feedback mechanism. The integrated protein secretion during the entire study period was 9.9 +/- 1.8 g in 120 min. The iv application of anti-GRP immunoglobulin caused a significant reduction to 5.0 +/- g in 120 min. No significant changes in the plasma concentrations of GRP and CCK were detectable. The increased secretion occurring after the administration of the protease inhibitor could be mediated by neural GRP-dependent mechanisms. These may also be relevant for CCK-dependent factors, which are only briefly mentioned. Topics: Animals; Cholecystokinin; Dogs; Esters; Feedback; Gabexate; Gastrin-Releasing Peptide; Guanidines; Pancreas; Peptides; Protease Inhibitors	1994
Gastrin-releasing peptide and CCK after intraduodenal inhibition of proteases. Annals of the New York Academy of Sciences, 1994, Mar-23, Volume: 713 Topics: Animals; Cholecystokinin; Dogs; Duodenum; Esters; Fistula; Gabexate; Gastrin-Releasing Peptide; Gastrins; Guanidines; Immunoglobulins; Infusions, Intravenous; Pancreas; Peptides; Protease Inhibitors; Proteins	1994

Article

Year

Gastrin-releasing peptide and CCK after intraduodenal inhibition of proteases in dogs.

International journal of pancreatology : official journal of the International Association of Pancreatology, 1994, Volume: 15, Issue:3

The intraduodenal application of a potent protease inhibitor (camostate, 600 mg) causes a significant increase in basal pancreatic secretion in the manner of a negative-feedback mechanism. The integrated protein secretion during the entire study period was 9.9 +/- 1.8 g in 120 min. The iv application of anti-GRP immunoglobulin caused a significant reduction to 5.0 +/- g in 120 min. No significant changes in the plasma concentrations of GRP and CCK were detectable. The increased secretion occurring after the administration of the protease inhibitor could be mediated by neural GRP-dependent mechanisms. These may also be relevant for CCK-dependent factors, which are only briefly mentioned.

Topics: Animals; Cholecystokinin; Dogs; Esters; Feedback; Gabexate; Gastrin-Releasing Peptide; Guanidines; Pancreas; Peptides; Protease Inhibitors

1994

Gastrin-releasing peptide and CCK after intraduodenal inhibition of proteases.

Annals of the New York Academy of Sciences, 1994, Mar-23, Volume: 713

Topics: Animals; Cholecystokinin; Dogs; Duodenum; Esters; Fistula; Gabexate; Gastrin-Releasing Peptide; Gastrins; Guanidines; Immunoglobulins; Infusions, Intravenous; Pancreas; Peptides; Protease Inhibitors; Proteins

1994