Page last updated: 2024-08-26

fulvestrant and ebselen

fulvestrant has been researched along with ebselen in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Hammock, BD; Hwang, SH; Morisseau, C; Newcomer, ME; Pakhomova, S	1
Felty, Q; Okoh, VO; Parkash, J; Poppiti, R; Roy, D	1

Other Studies

2 other study(ies) available for fulvestrant and ebselen

Article	Year
Inhibition of soluble epoxide hydrolase by fulvestrant and sulfoxides. Bioorganic & medicinal chemistry letters, 2013, Jul-01, Volume: 23, Issue:13 Topics: Dose-Response Relationship, Drug; Enzyme Inhibitors; Epoxide Hydrolases; Estradiol; Fulvestrant; Humans; Molecular Structure; Solubility; Structure-Activity Relationship; Sulfoxides	2013
Reactive oxygen species via redox signaling to PI3K/AKT pathway contribute to the malignant growth of 4-hydroxy estradiol-transformed mammary epithelial cells. PloS one, 2013, Volume: 8, Issue:2 Topics: Animals; Azoles; Catalase; Catechols; Cell Cycle Proteins; Cell Proliferation; Cell Transformation, Neoplastic; Collagen; Colony-Forming Units Assay; Dose-Response Relationship, Drug; Epithelial Cells; Estradiol; Estrogens, Catechol; Fulvestrant; Gene Expression Regulation, Neoplastic; Humans; Isoindoles; Mammary Glands, Human; Mice; Models, Biological; Neoplasm Invasiveness; Organoselenium Compounds; Oxidation-Reduction; Phenotype; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Reactive Oxygen Species; Signal Transduction; Spheroids, Cellular	2013

Article

Year

Inhibition of soluble epoxide hydrolase by fulvestrant and sulfoxides.

Bioorganic & medicinal chemistry letters, 2013, Jul-01, Volume: 23, Issue:13

Topics: Dose-Response Relationship, Drug; Enzyme Inhibitors; Epoxide Hydrolases; Estradiol; Fulvestrant; Humans; Molecular Structure; Solubility; Structure-Activity Relationship; Sulfoxides

2013

Reactive oxygen species via redox signaling to PI3K/AKT pathway contribute to the malignant growth of 4-hydroxy estradiol-transformed mammary epithelial cells.

PloS one, 2013, Volume: 8, Issue:2

Topics: Animals; Azoles; Catalase; Catechols; Cell Cycle Proteins; Cell Proliferation; Cell Transformation, Neoplastic; Collagen; Colony-Forming Units Assay; Dose-Response Relationship, Drug; Epithelial Cells; Estradiol; Estrogens, Catechol; Fulvestrant; Gene Expression Regulation, Neoplastic; Humans; Isoindoles; Mammary Glands, Human; Mice; Models, Biological; Neoplasm Invasiveness; Organoselenium Compounds; Oxidation-Reduction; Phenotype; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Reactive Oxygen Species; Signal Transduction; Spheroids, Cellular

2013