fpl 64176 has been researched along with calpain in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, X; Dore, J; Liu, Y | 1 |
| Banik, NL; Del Re, AM; Ray, SK; Sribnick, EA; Woodward, JJ | 1 |
2 other study(ies) available for fpl 64176 and calpain
| Article | Year |
|---|---|
Calcium influx through L-type channels generates protein kinase M to induce burst firing of dopamine cells in the rat ventral tegmental area.
Topics: 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; Animals; Calcium; Calcium Channel Agonists; Calcium Channels, L-Type; Calpain; Dopamine; Female; Glutamine; Protein Kinase C; Pyrroles; Rats; Rats, Sprague-Dawley; Receptors, Cholinergic; Ventral Tegmental Area | 2007 |
Estrogen attenuates glutamate-induced cell death by inhibiting Ca2+ influx through L-type voltage-gated Ca2+ channels.
Topics: Animals; Apoptosis; Calcium; Calcium Channel Agonists; Calcium Channels, L-Type; Calpain; Caspase 3; Cell Line; Cell Physiological Phenomena; Electric Capacitance; Estradiol; Estrogens; Glutamic Acid; Intracellular Space; Membrane Potentials; Mice; Neurons; Neuroprotective Agents; Pyrroles; Rats | 2009 |