fluoxetine and n-(1-methyl-5-indolyl)-n'-(3-methyl-5-isothiazolyl)urea

fluoxetine has been researched along with n-(1-methyl-5-indolyl)-n'-(3-methyl-5-isothiazolyl)urea in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	2 (100.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Cai, L; Hertz, L; Li, B; Nu, W; Peng, L; Zhang, H; Zhang, S	1
Hertz, L; Li, B; Li, M; Peng, L; Zhang, S	1

Other Studies

2 other study(ies) available for fluoxetine and n-(1-methyl-5-indolyl)-n'-(3-methyl-5-isothiazolyl)urea

Article	Year
Fluoxetine-mediated 5-HT2B receptor stimulation in astrocytes causes EGF receptor transactivation and ERK phosphorylation. Psychopharmacology, 2008, Volume: 201, Issue:3 Topics: Aminopyridines; Animals; Antidepressive Agents, Second-Generation; Astrocytes; Butadienes; Calcium; Cells, Cultured; Chelating Agents; Dipeptides; Dose-Response Relationship, Drug; Egtazic Acid; ErbB Receptors; Extracellular Signal-Regulated MAP Kinases; Fluorobenzenes; Fluoxetine; Gene Expression; Indoles; Maleimides; Mice; Nitriles; Phosphorylation; Piperidines; Protein Kinase C; Proto-Oncogene Proteins c-fos; Quinazolines; Receptor, Serotonin, 5-HT2B; RNA, Messenger; Signal Transduction; Substrate Specificity; Transcriptional Activation; Tyrphostins; Up-Regulation; Urea	2008
Chronic treatment of astrocytes with therapeutically relevant fluoxetine concentrations enhances cPLA2 expression secondary to 5-HT2B-induced, transactivation-mediated ERK1/2 phosphorylation. Psychopharmacology, 2009, Volume: 207, Issue:1 Topics: Animals; Astrocytes; Cells, Cultured; Cerebral Cortex; Dose-Response Relationship, Drug; Enzyme Inhibitors; Fluoxetine; Gene Expression Regulation; Indoles; Mice; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Phospholipases A2; Phosphorylation; Receptor, Serotonin, 5-HT2B; RNA, Messenger; Selective Serotonin Reuptake Inhibitors; Serotonin 5-HT2 Receptor Antagonists; Time Factors; Urea	2009

Article

Year

Fluoxetine-mediated 5-HT2B receptor stimulation in astrocytes causes EGF receptor transactivation and ERK phosphorylation.

Psychopharmacology, 2008, Volume: 201, Issue:3

Topics: Aminopyridines; Animals; Antidepressive Agents, Second-Generation; Astrocytes; Butadienes; Calcium; Cells, Cultured; Chelating Agents; Dipeptides; Dose-Response Relationship, Drug; Egtazic Acid; ErbB Receptors; Extracellular Signal-Regulated MAP Kinases; Fluorobenzenes; Fluoxetine; Gene Expression; Indoles; Maleimides; Mice; Nitriles; Phosphorylation; Piperidines; Protein Kinase C; Proto-Oncogene Proteins c-fos; Quinazolines; Receptor, Serotonin, 5-HT2B; RNA, Messenger; Signal Transduction; Substrate Specificity; Transcriptional Activation; Tyrphostins; Up-Regulation; Urea

2008

Chronic treatment of astrocytes with therapeutically relevant fluoxetine concentrations enhances cPLA2 expression secondary to 5-HT2B-induced, transactivation-mediated ERK1/2 phosphorylation.

Psychopharmacology, 2009, Volume: 207, Issue:1

Topics: Animals; Astrocytes; Cells, Cultured; Cerebral Cortex; Dose-Response Relationship, Drug; Enzyme Inhibitors; Fluoxetine; Gene Expression Regulation; Indoles; Mice; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Phospholipases A2; Phosphorylation; Receptor, Serotonin, 5-HT2B; RNA, Messenger; Selective Serotonin Reuptake Inhibitors; Serotonin 5-HT2 Receptor Antagonists; Time Factors; Urea

2009