fluorouracil and trichostatin a

fluorouracil has been researched along with trichostatin a in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (16.67)18.2507
2000's3 (50.00)29.6817
2010's2 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Ando, T; Mariko, Y; Nakanishi, O; Nosaka, Y; Saito, A; Suzuki, T; Tsuchiya, K; Tsuruo, T; Yamashita, T1
Kitajima, Y; Koga, Y; Miyazaki, K; Miyoshi, A; Sato, K1
Hirakawa, K; Ren, J; Yashiro, M; Zhang, X1
Bang, YJ; Jong, HS; Jung, Y; Kim, JH; Kim, TY; Lee, JH; Park, JH1
Damante, G; Di Loreto, C; Pandolfi, M; Puglisi, F; Puppin, C1
Choi, HG; Jeon, CK; Kang, HR; Kim, SH; Lim, SJ1

Other Studies

6 other study(ies) available for fluorouracil and trichostatin a

ArticleYear
A synthetic inhibitor of histone deacetylase, MS-27-275, with marked in vivo antitumor activity against human tumors.
    Proceedings of the National Academy of Sciences of the United States of America, 1999, Apr-13, Volume: 96, Issue:8

    Topics: Animals; Antineoplastic Agents; Benzamides; Butyrates; Cell Cycle; Cell Survival; Colonic Neoplasms; Cyclin-Dependent Kinase Inhibitor p21; Cyclins; Enzyme Inhibitors; Female; Fluorouracil; Gelsolin; Histone Deacetylase Inhibitors; HL-60 Cells; Humans; Hydroxamic Acids; K562 Cells; KB Cells; Mice; Mice, Nude; Ovarian Neoplasms; Pancreatic Neoplasms; Pyridines; Transplantation, Heterologous; Tumor Cells, Cultured

1999
Deficient expression of the DPD gene is caused by epigenetic modification in biliary tract cancer cells, and induces high sensitivity to 5-FU treatment.
    International journal of oncology, 2006, Volume: 29, Issue:2

    Topics: Antimetabolites, Antineoplastic; Base Sequence; Biliary Tract Neoplasms; Cell Line, Tumor; Enzyme Inhibitors; Epigenesis, Genetic; Fluorouracil; Gene Expression Regulation, Neoplastic; Histone Deacetylase Inhibitors; Humans; Hydroxamic Acids; Molecular Sequence Data; Protein Synthesis Inhibitors; Tetrazolium Salts; Thiazoles

2006
Histone deacetylase inhibitor, trichostatin A, increases the chemosensitivity of anticancer drugs in gastric cancer cell lines.
    Oncology reports, 2006, Volume: 16, Issue:3

    Topics: Antineoplastic Agents; Apoptosis; Camptothecin; Cell Proliferation; Deoxycytidine; Drug Resistance, Neoplasm; Drug Synergism; Drug Therapy, Combination; Enzyme Inhibitors; Fluorouracil; Gemcitabine; Histone Deacetylase Inhibitors; Humans; Hydroxamic Acids; Irinotecan; Organoplatinum Compounds; Oxaliplatin; Paclitaxel; Stomach Neoplasms; Tumor Cells, Cultured

2006
Histone deacetylase inhibitor enhances 5-fluorouracil cytotoxicity by down-regulating thymidylate synthase in human cancer cells.
    Molecular cancer therapeutics, 2006, Volume: 5, Issue:12

    Topics: Acetylation; Antineoplastic Combined Chemotherapy Protocols; Cell Line, Tumor; Cycloheximide; Dose-Response Relationship, Drug; Down-Regulation; Drug Resistance, Neoplasm; Drug Synergism; Fluorouracil; Histone Deacetylase Inhibitors; Histone Deacetylases; HSP70 Heat-Shock Proteins; HSP90 Heat-Shock Proteins; Humans; Hydroxamic Acids; Neoplasms; Proteasome Endopeptidase Complex; RNA, Messenger; Thymidylate Synthase

2006
Histone deacetylase inhibitors induce thymidine phosphorylase expression in cultured breast cancer cell lines.
    Oncology reports, 2011, Volume: 26, Issue:2

    Topics: Breast Neoplasms; Capecitabine; Cell Line, Tumor; Cell Survival; Deoxycytidine; Drug Synergism; Female; Fluorouracil; Histone Deacetylase Inhibitors; Humans; Hydroxamic Acids; Immunohistochemistry; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Thymidine Phosphorylase; Vorinostat

2011
Butyrate-mediated acquisition of chemoresistance by human colon cancer cells.
    Oncology reports, 2016, Volume: 36, Issue:2

    Topics: Apoptosis; ATP Binding Cassette Transporter, Subfamily B, Member 1; ATP Binding Cassette Transporter, Subfamily G, Member 2; bcl-X Protein; Butyrates; Cell Line, Tumor; Cell Movement; Cell Proliferation; Colonic Neoplasms; Doxorubicin; Drug Resistance, Neoplasm; Fluorouracil; HCT116 Cells; Humans; Hydroxamic Acids; Multidrug Resistance-Associated Proteins; Neoplasm Invasiveness; Neoplasm Proteins; Paclitaxel

2016