etoposide has been researched along with pyrazolanthrone in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 5 (83.33) | 29.6817 |
| 2010's | 1 (16.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bellows, DS; Clarke, ID; Diamandis, P; Dirks, PB; Graham, J; Jamieson, LG; Ling, EK; Sacher, AG; Tyers, M; Ward, RJ; Wildenhain, J | 1 |
| Austin, CP; Fidock, DA; Hayton, K; Huang, R; Inglese, J; Jiang, H; Johnson, RL; Su, XZ; Wellems, TE; Wichterman, J; Yuan, J | 1 |
| Barnes, JC; Bradley, P; Day, NC; Fourches, D; Reed, JZ; Tropsha, A | 1 |
| Besirli, CG; Johnson, EM | 1 |
| Broaddus, VC; Liu, W; Vivo, C | 1 |
| Deng, R; Feng, GK; Guan, Z; Han, Y; Huang, W; Li, MT; Li, W; Liu, ZC; Mei, YP; Wang, Y; Zeng, YX; Zhou, JM; Zhu, XF | 1 |
6 other study(ies) available for etoposide and pyrazolanthrone
| Article | Year |
|---|---|
Chemical genetics reveals a complex functional ground state of neural stem cells.
Topics: Animals; Cell Survival; Cells, Cultured; Mice; Molecular Structure; Neoplasms; Neurons; Pharmaceutical Preparations; Sensitivity and Specificity; Stem Cells | 2007 |
Genetic mapping of targets mediating differential chemical phenotypes in Plasmodium falciparum.
Topics: Animals; Antimalarials; ATP Binding Cassette Transporter, Subfamily B, Member 1; Chromosome Mapping; Crosses, Genetic; Dihydroergotamine; Drug Design; Drug Resistance; Humans; Inhibitory Concentration 50; Mutation; Plasmodium falciparum; Quantitative Trait Loci; Transfection | 2009 |
Cheminformatics analysis of assertions mined from literature that describe drug-induced liver injury in different species.
Topics: Animals; Chemical and Drug Induced Liver Injury; Cluster Analysis; Databases, Factual; Humans; MEDLINE; Mice; Models, Chemical; Molecular Conformation; Quantitative Structure-Activity Relationship | 2010 |
JNK-independent activation of c-Jun during neuronal apoptosis induced by multiple DNA-damaging agents.
Topics: Animals; Animals, Newborn; Anthracenes; Apoptosis; Carbazoles; Cell Culture Techniques; Cell Survival; Cytarabine; DNA; DNA Damage; Enzyme Inhibitors; Etoposide; Indoles; JNK Mitogen-Activated Protein Kinases; Kinetics; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Nerve Growth Factor; Neurons; Proto-Oncogene Proteins c-jun; Rats; Rats, Sprague-Dawley; Superior Cervical Ganglion; Transcription Factor AP-1 | 2003 |
c-Jun N-terminal kinase contributes to apoptotic synergy induced by tumor necrosis factor-related apoptosis-inducing ligand plus DNA damage in chemoresistant, p53 inactive mesothelioma cells.
Topics: Anthracenes; Apoptosis; Apoptosis Regulatory Proteins; Blotting, Western; Caspase 9; Caspases; Cell Survival; DNA Damage; Dose-Response Relationship, Radiation; Drug Resistance, Neoplasm; Enzyme Activation; Enzyme Inhibitors; Etoposide; Genes, Dominant; Genes, p53; Humans; JNK Mitogen-Activated Protein Kinases; Lung Neoplasms; Membrane Glycoproteins; Mesothelioma; Mitogen-Activated Protein Kinase 8; Mitogen-Activated Protein Kinase 9; Mitogen-Activated Protein Kinases; Mutation; Phosphorylation; Recombinant Proteins; Time Factors; TNF-Related Apoptosis-Inducing Ligand; Transfection; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha; Tumor Suppressor Protein p53; Ultraviolet Rays | 2003 |
Acetylcholinesterase expression mediated by c-Jun-NH2-terminal kinase pathway during anticancer drug-induced apoptosis.
Topics: Acetylcholinesterase; Adenoviridae; Anthracenes; Antineoplastic Agents; Apoptosis; Caspase 3; Cell Line, Tumor; Cytoprotection; Diterpenes; Enzyme Activation; Etoposide; Gene Expression Regulation, Neoplastic; Humans; JNK Mitogen-Activated Protein Kinases; Poly(ADP-ribose) Polymerases; RNA, Small Interfering; Signal Transduction; Up-Regulation | 2006 |