Page last updated: 2024-08-23

etoposide and pepstatin

etoposide has been researched along with pepstatin in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (25.00)18.2507
2000's2 (50.00)29.6817
2010's0 (0.00)24.3611
2020's1 (25.00)2.80

Authors

AuthorsStudies
Anderson, E; Ekins, S; Foil, DH; Havener, TM; Hickey, AJ; Makarov, V; Monakhova, N; Puhl, AC; Rank, L; Riabova, O; Zorn, KM1
El-Deiry, WS; Peters, C; Saftig, P; Wu, GS1
Choi, YK; Chung, YG; Jeon, HM; Jin, X; Jung, JE; Kim, H; Kim, TK; Lee, JB; Lee, JS; Oh, SY; Park, HJ; Park, JW; Sohn, YW; You, S1
Beaujouin, M; Liaudet-Coopman, E1

Other Studies

4 other study(ies) available for etoposide and pepstatin

ArticleYear
Multiple approaches to repurposing drugs for neuroblastoma.
    Bioorganic & medicinal chemistry, 2022, 11-01, Volume: 73

    Topics: Bayes Theorem; Cell Line, Tumor; Child; Child, Preschool; Crizotinib; Drug Repositioning; Etoposide; Fingolimod Hydrochloride; Humans; Neuroblastoma; Niclosamide

2022
Potential role for cathepsin D in p53-dependent tumor suppression and chemosensitivity.
    Oncogene, 1998, Apr-30, Volume: 16, Issue:17

    Topics: Animals; Apoptosis; Cathepsin D; Cell Line; Colonic Neoplasms; DNA Damage; Doxorubicin; Drug Resistance, Neoplasm; Etoposide; Female; Fibroblasts; Genes, p53; Humans; Mice; Ovarian Neoplasms; Pepstatins; RNA, Messenger; Transcriptional Activation; Tumor Cells, Cultured; Up-Regulation

1998
Selective cell death of oncogenic Akt-transduced brain cancer cells by etoposide through reactive oxygen species mediated damage.
    Molecular cancer therapeutics, 2007, Volume: 6, Issue:8

    Topics: Animals; Antineoplastic Agents; Astrocytes; Brain Neoplasms; Cell Death; Cell Transformation, Neoplastic; Cyclin-Dependent Kinase Inhibitor p16; Dose-Response Relationship, Drug; Etoposide; Mice; Mice, Nude; Oncogene Protein v-akt; Pepstatins; Reactive Oxygen Species; Time Factors; Transduction, Genetic; Tumor Suppressor Protein p53

2007
Cathepsin D overexpressed by cancer cells can enhance apoptosis-dependent chemo-sensitivity independently of its catalytic activity.
    Advances in experimental medicine and biology, 2008, Volume: 617

    Topics: Antineoplastic Agents, Phytogenic; Apoptosis; Caspase 3; Caspase 9; Catalysis; Cathepsin D; Cytochromes c; Cytosol; Drug Resistance, Neoplasm; Etoposide; Humans; Neoplasms; Pepstatins; Protease Inhibitors; Tumor Cells, Cultured

2008