erythrosine has been researched along with tiopronin in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (50.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Becker, LB; Li, C; Schumacker, PT; Shao, Z; Vanden Hoek, TL | 1 |
| Abdallah, Y; Erdogan, A; Gündüz, D; Iraqi, W; Kasseckert, SA; Ladilov, Y; Neuhof, C; Piper, HM; Reusch, HP; Said, M; Schlüter, KD; Shahzad, T; Tillmanns, H | 1 |
2 other study(ies) available for erythrosine and tiopronin
| Article | Year |
|---|---|
Mitochondrial electron transport can become a significant source of oxidative injury in cardiomyocytes.
Topics: Animals; Antioxidants; Biological Transport; Cell Death; Cell Survival; Cells, Cultured; Chick Embryo; Cyanides; Electrons; Fluoresceins; Mitochondria, Heart; Myocardial Contraction; Myocardium; Oxidants; Oxidative Stress; Oxygen; Phenanthrolines; Tiopronin | 1997 |
Interplay between Ca2+ cycling and mitochondrial permeability transition pores promotes reperfusion-induced injury of cardiac myocytes.
Topics: Acetylcysteine; Adenosine Triphosphate; Animals; Calcium; Cyclosporine; Fluoresceins; Male; Membrane Potential, Mitochondrial; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Reperfusion Injury; Myocytes, Cardiac; Necrosis; Rats; Rats, Wistar; Reactive Oxygen Species; Ruthenium Compounds; Ryanodine; Thapsigargin; Tiopronin | 2011 |