erlotinib hydrochloride has been researched along with oleanolic acid in 1 studies
Studies (erlotinib hydrochloride) | Trials (erlotinib hydrochloride) | Recent Studies (post-2010) (erlotinib hydrochloride) | Studies (oleanolic acid) | Trials (oleanolic acid) | Recent Studies (post-2010) (oleanolic acid) |
---|---|---|---|---|---|
4,353 | 786 | 3,033 | 4,169 | 31 | 2,404 |
Protein | Taxonomy | erlotinib hydrochloride (IC50) | oleanolic acid (IC50) |
---|---|---|---|
Aldo-keto reductase family 1 member B10 | Homo sapiens (human) | 2.045 | |
Phospholipase A2 | Homo sapiens (human) | 3 | |
DNA polymerase beta | Homo sapiens (human) | 6.8667 | |
DNA polymerase beta | Rattus norvegicus (Norway rat) | 5.6 | |
Receptor-type tyrosine-protein phosphatase C | Homo sapiens (human) | 1 | |
Receptor-type tyrosine-protein phosphatase F | Homo sapiens (human) | 3.065 | |
Tissue factor | Homo sapiens (human) | 0.005 | |
Tyrosine-protein phosphatase non-receptor type 2 | Homo sapiens (human) | 4.38 | |
Tyrosine-protein phosphatase non-receptor type 1 | Homo sapiens (human) | 2.8444 | |
Liver carboxylesterase 1 | Homo sapiens (human) | 0.1 | |
Tyrosine-protein phosphatase non-receptor type 6 | Homo sapiens (human) | 3.065 | |
M-phase inducer phosphatase 2 | Homo sapiens (human) | 0.98 | |
Dual specificity protein phosphatase 3 | Homo sapiens (human) | 5.4 | |
Transcription factor p65 | Homo sapiens (human) | 2.4 | |
Tyrosine-protein phosphatase non-receptor type 11 | Homo sapiens (human) | 3.065 | |
Protease | Human immunodeficiency virus 1 | 8 |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (100.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Black, CC; Dmitrovsky, E; Gribble, GW; Honda, T; Lamph, WW; Liby, K; Liu, X; Risingsong, R; Royce, DB; Sporn, MB; Sporn, TA; Williams, CR; Yore, MM | 1 |
1 other study(ies) available for erlotinib hydrochloride and oleanolic acid
Article | Year |
---|---|
The rexinoid LG100268 and the synthetic triterpenoid CDDO-methyl amide are more potent than erlotinib for prevention of mouse lung carcinogenesis.
Topics: Animals; Anticarcinogenic Agents; Cell Line, Tumor; Erlotinib Hydrochloride; Female; Lung; Lung Neoplasms; Mice; Nicotinic Acids; Oleanolic Acid; Protein Kinase Inhibitors; Quinazolines; Tetrahydronaphthalenes | 2008 |