erlotinib hydrochloride has been researched along with bisindolylmaleimide i in 1 studies
Studies (erlotinib hydrochloride) | Trials (erlotinib hydrochloride) | Recent Studies (post-2010) (erlotinib hydrochloride) | Studies (bisindolylmaleimide i) | Trials (bisindolylmaleimide i) | Recent Studies (post-2010) (bisindolylmaleimide i) |
---|---|---|---|---|---|
4,353 | 786 | 3,033 | 1,198 | 1 | 174 |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Ballestrero, A; Bauer, I; Castiglioni, I; D'Agostino, V; Feldmann, G; Latorre, E; Nencioni, A; Pancher, M; Provenzani, A; Thongon, N; Zucal, C | 1 |
1 other study(ies) available for erlotinib hydrochloride and bisindolylmaleimide i
Article | Year |
---|---|
The GSK3β inhibitor BIS I reverts YAP-dependent EMT signature in PDAC cell lines by decreasing SMADs expression level.
Topics: Adaptor Proteins, Signal Transducing; Antineoplastic Agents; Carcinoma, Pancreatic Ductal; Cell Line, Tumor; Drug Screening Assays, Antitumor; Epithelial-Mesenchymal Transition; Erlotinib Hydrochloride; Gene Expression Regulation, Neoplastic; Glycogen Synthase Kinase 3 beta; Humans; Indoles; Maleimides; Pancreatic Neoplasms; Phosphoproteins; Smad Proteins; Transcription Factors; YAP-Signaling Proteins | 2016 |