divicine has been researched along with ascorbic acid in 8 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 8 (100.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Benatti, U; De Flora, A; Guida, L | 1 |
Ciriolo, MR; Mavelli, I; Rotilio, G | 1 |
Benatti, U; De Flora, A; Guida, L; Morelli, A | 2 |
Arese, P; Baker, MA; Bosia, A; Pescarmona, G; Turrini, F | 1 |
Chevion, M; Navok, T | 1 |
Benatti, U; Ciriolo, MR; De Flora, A; Forteleoni, G; Mavelli, I; Meloni, T; Morelli, A; Rossi, L; Rotilio, G | 1 |
Arese, P; Bosia, A; D'Aquino, M; Gaetani, GF; Gaetani, S; Naitana, A | 1 |
8 other study(ies) available for divicine and ascorbic acid
Article | Year |
---|---|
The role of plasma in oxidative haemolysis.
Topics: Ascorbic Acid; Carbon Monoxide; Erythrocytes; Glucosephosphate Dehydrogenase Deficiency; Hemolysis; Humans; Oxidation-Reduction; Peroxides; Plasma; Pyrimidinones; tert-Butylhydroperoxide | 1986 |
Effect of the redox state of the red blood cell components on the inactivation of glutathione peroxidase by divicine.
Topics: Ascorbic Acid; Catalase; Cell-Free System; Erythrocytes; Glutathione; Glutathione Peroxidase; Humans; In Vitro Techniques; Methemoglobin; NADP; Oxidation-Reduction; Pyrimidinones | 1986 |
Oxidant damage of normal and glucose 6-phosphate dehydrogenase (G6PD)-deficient red blood cells is enhanced by iron-EDTA complex.
Topics: Ascorbic Acid; Catalase; Edetic Acid; Erythrocytes; Ferric Compounds; Free Radicals; Glucosephosphate Dehydrogenase Deficiency; Glutathione; Humans; Hydroxides; Hydroxyl Radical; Iron; Male; Oxygen; Pyrimidinones | 1983 |
Mechanism of action of divicine in a cell-free system and in glucose-6-phosphate dehydrogenase-deficient red cells.
Topics: Ascorbic Acid; Cell-Free System; Cytochrome c Group; Erythrocytes; Favism; Glucosephosphate Dehydrogenase Deficiency; Glutathione; Hemolysis; Humans; Hydrogen Peroxide; In Vitro Techniques; Kinetics; Male; Oxidation-Reduction; Pyrimidinones; Superoxides | 1984 |
Transition metals mediate enzymatic inactivation caused by favism-inducing agents.
Topics: Ascorbic Acid; Barbiturates; Cholinesterase Inhibitors; Copper; Electron Spin Resonance Spectroscopy; Favism; Humans; In Vitro Techniques; Oxidation-Reduction; Pyrimidinones | 1984 |
Mediterranean glucose 6-phosphate dehydrogenase (G6PD) deficiency--near normal decay of the mutant enzyme protein in circulating erythrocytes.
Topics: Ascorbic Acid; Erythrocyte Aging; Erythrocytes; Glucosephosphate Dehydrogenase; Glucosephosphate Dehydrogenase Deficiency; Hemolysis; Humans; Male; Mediterranean Islands; Mutation; Pyrimidinones | 1984 |
Favism: a hemolytic disease associated with increased superoxide dismutase and decreased glutathione peroxidase activities in red blood cells.
Topics: Ascorbic Acid; Electrophoresis, Polyacrylamide Gel; Erythrocyte Aging; Erythrocytes; Favism; Glutathione Peroxidase; Hemolysis; Humans; Oxidation-Reduction; Pyrimidinones; Reticulocytes; Superoxide Dismutase | 1984 |
Effect of divicine and isouramil on red cell metabolism in normal and G6PD-deficient (Mediterranean variant) subjects. Possible role in the genesis of favism.
Topics: Ascorbic Acid; Barbiturates; Erythrocytes; Favism; Glucosephosphate Dehydrogenase Deficiency; Glucosides; Glutathione; Humans; Kinetics; Pyrimidinones; Reference Values; Species Specificity; Toxins, Biological; Uracil; Uridine | 1981 |