Page last updated: 2024-08-21

diphenylamine and navitoclax

diphenylamine has been researched along with navitoclax in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Cha, HJ; Go, YH; Hong, SK; Kwon, OS; Kwon, SJ; Oh, E	1
Archibald, HL; Azzoli, CG; Benes, CH; Bilton, SJ; Brown, SP; Caenepeel, S; Cahill, DP; Coxon, A; Farago, AF; Ferris, LA; Fraser, C; Gainor, JF; Gomez-Caraballo, M; Hata, AN; Hughes, PE; Keyes, C; Lanuti, M; Li, C; Nangia, V; Phan, N; Piotrowska, Z; Raskin, KA; Rigas, D; Sarosiek, KA; Sequist, LV; Shin, JH; Siddiqui, FM; Timonina, D; Vajda, K; Wright, CD	1

Other Studies

2 other study(ies) available for diphenylamine and navitoclax

Article	Year
BCL2 induced by LAMTOR3/MAPK is a druggable target of chemoradioresistance in mesenchymal lung cancer. Cancer letters, 2017, 09-10, Volume: 403 Topics: A549 Cells; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenocarcinoma of Lung; Aniline Compounds; Antineoplastic Combined Chemotherapy Protocols; Benzamides; Biphenyl Compounds; Cell Proliferation; Cell Survival; Chemoradiotherapy; Diphenylamine; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Etoposide; Gene Expression Regulation, Neoplastic; Humans; Lung Neoplasms; MAP Kinase Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Molecular Mimicry; Nitrophenols; Peptide Fragments; Piperazines; Protein Kinase Inhibitors; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-bcl-2; Radiation Tolerance; RNA Interference; Signal Transduction; Sulfonamides; Transfection; Up-Regulation	2017
Exploiting MCL1 Dependency with Combination MEK + MCL1 Inhibitors Leads to Induction of Apoptosis and Tumor Regression in Cancer discovery, 2018, Volume: 8, Issue:12 Topics: A549 Cells; Aniline Compounds; Animals; Antineoplastic Agents; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Benzamides; Carcinoma, Non-Small-Cell Lung; Cell Line, Tumor; Diphenylamine; Humans; Lung Neoplasms; Mice, Knockout; Mice, Nude; Mice, SCID; Mitogen-Activated Protein Kinase Kinases; Myeloid Cell Leukemia Sequence 1 Protein; Protein Kinase Inhibitors; Proto-Oncogene Proteins p21(ras); Sulfonamides; Tumor Burden; Xenograft Model Antitumor Assays	2018

Article

Year

BCL2 induced by LAMTOR3/MAPK is a druggable target of chemoradioresistance in mesenchymal lung cancer.

Cancer letters, 2017, 09-10, Volume: 403

Topics: A549 Cells; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenocarcinoma of Lung; Aniline Compounds; Antineoplastic Combined Chemotherapy Protocols; Benzamides; Biphenyl Compounds; Cell Proliferation; Cell Survival; Chemoradiotherapy; Diphenylamine; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Etoposide; Gene Expression Regulation, Neoplastic; Humans; Lung Neoplasms; MAP Kinase Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Molecular Mimicry; Nitrophenols; Peptide Fragments; Piperazines; Protein Kinase Inhibitors; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-bcl-2; Radiation Tolerance; RNA Interference; Signal Transduction; Sulfonamides; Transfection; Up-Regulation

2017

Exploiting MCL1 Dependency with Combination MEK + MCL1 Inhibitors Leads to Induction of Apoptosis and Tumor Regression in

Cancer discovery, 2018, Volume: 8, Issue:12

Topics: A549 Cells; Aniline Compounds; Animals; Antineoplastic Agents; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Benzamides; Carcinoma, Non-Small-Cell Lung; Cell Line, Tumor; Diphenylamine; Humans; Lung Neoplasms; Mice, Knockout; Mice, Nude; Mice, SCID; Mitogen-Activated Protein Kinase Kinases; Myeloid Cell Leukemia Sequence 1 Protein; Protein Kinase Inhibitors; Proto-Oncogene Proteins p21(ras); Sulfonamides; Tumor Burden; Xenograft Model Antitumor Assays

2018