diphenylamine has been researched along with abt-737 in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Andreeff, M; Bergamo, P; Bornmann, W; Chen, W; Konopleva, M; Korchin, B; Mak, DH; McCubrey, J; McQueen, T; Milella, M; Ricciardi, MR; Ruvolo, P; Tafuri, A; Tsao, T; Watts, JC | 1 |
Amin, S; Brauner, E; Gunda, V; Kim, YS; Letai, A; Parangi, S; Sarosiek, KA; Zhou, Z | 1 |
Cha, HJ; Go, YH; Hong, SK; Kwon, OS; Kwon, SJ; Oh, E | 1 |
3 other study(ies) available for diphenylamine and abt-737
Article | Year |
---|---|
MEK inhibition enhances ABT-737-induced leukemia cell apoptosis via prevention of ERK-activated MCL-1 induction and modulation of MCL-1/BIM complex.
Topics: Animals; Antineoplastic Agents; Apoptosis; Apoptosis Regulatory Proteins; bcl-2 Homologous Antagonist-Killer Protein; bcl-2-Associated X Protein; Bcl-2-Like Protein 11; Benzamides; Biphenyl Compounds; Cell Line, Tumor; Diphenylamine; Extracellular Signal-Regulated MAP Kinases; Humans; Leukemia, Myeloid, Acute; Membrane Proteins; Mice; Mice, Nude; Mitogen-Activated Protein Kinase Kinases; Myeloid Cell Leukemia Sequence 1 Protein; Nitrophenols; Piperazines; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-bcl-2; Sulfonamides | 2012 |
Inhibition of MAPKinase pathway sensitizes thyroid cancer cells to ABT-737 induced apoptosis.
Topics: Animals; Apoptosis; bcl-X Protein; Benzamides; Biphenyl Compounds; Cell Line, Tumor; Diphenylamine; Female; Humans; Indoles; MAP Kinase Signaling System; Mice; Mitogen-Activated Protein Kinases; Nitrophenols; Piperazines; Proto-Oncogene Proteins c-bcl-2; Sulfonamides; Thyroid Neoplasms | 2017 |
BCL2 induced by LAMTOR3/MAPK is a druggable target of chemoradioresistance in mesenchymal lung cancer.
Topics: A549 Cells; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenocarcinoma of Lung; Aniline Compounds; Antineoplastic Combined Chemotherapy Protocols; Benzamides; Biphenyl Compounds; Cell Proliferation; Cell Survival; Chemoradiotherapy; Diphenylamine; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Etoposide; Gene Expression Regulation, Neoplastic; Humans; Lung Neoplasms; MAP Kinase Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Molecular Mimicry; Nitrophenols; Peptide Fragments; Piperazines; Protein Kinase Inhibitors; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-bcl-2; Radiation Tolerance; RNA Interference; Signal Transduction; Sulfonamides; Transfection; Up-Regulation | 2017 |