dicarbethoxydihydrocollidine has been researched along with cholic acid in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 3 (75.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Denk, H; Fickert, P; Fuchsbichler, A; Stumptner, C; Trauner, M; Zatloukal, K | 1 |
| Desai, MS; Felix, JC; Karpen, SJ; Kosters, A | 1 |
| Desai, MS; Eblimit, Z; Karpen, SJ; Kosters, A; Moore, DD; Penny, DJ; Thevananther, S | 1 |
| Chen, Y; Fickert, P; Gassler, N; Guldiken, N; Haybaeck, J; Jeon, MK; Mohammed, HH; Pollheimer, MJ; Spurny, M; Strnad, P; Trautwein, C | 1 |
4 other study(ies) available for dicarbethoxydihydrocollidine and cholic acid
| Article | Year |
|---|---|
Bile acid-induced Mallory body formation in drug-primed mouse liver.
Topics: Animals; Bile Acids and Salts; Bile Ducts; Cholestasis; Cholic Acid; Dicarbethoxydihydrocollidine; Diet; Hepatocytes; Humans; Inclusion Bodies; Keratins; Ligation; Liver; Male; Mice; Phosphorylation; Ubiquitin | 2002 |
Impaired bile acid handling and aggravated liver injury in mice expressing a hepatocyte-specific RXRα variant lacking the DNA-binding domain.
Topics: Animals; Apoptosis; Bile Acids and Salts; Cholestasis; Cholic Acid; Gene Expression; Hepatocytes; Liver; Male; Mice; Mice, 129 Strain; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Knockout; Mice, Mutant Strains; Mutant Proteins; Peptide Fragments; Protein Structure, Tertiary; Pyridines; Retinoid X Receptor alpha | 2014 |
Cardiomyopathy reverses with recovery of liver injury, cholestasis and cholanemia in mouse model of biliary fibrosis.
Topics: Adrenergic beta-Agonists; Animals; ATP Binding Cassette Transporter, Subfamily B, Member 11; ATP-Binding Cassette Transporters; Biomarkers; Cardiomyopathies; Cholestasis; Cholic Acid; Disease Models, Animal; Drug Resistance; Liver; Liver Cirrhosis, Biliary; Male; Mice, Inbred C57BL; Mice, Knockout; Pyridines; Recovery of Function; Signal Transduction; Time Factors; Ventricular Function, Left | 2015 |
Loss of keratin 19 favours the development of cholestatic liver disease through decreased ductular reaction.
Topics: Animals; Cell Proliferation; Chemical and Drug Induced Liver Injury; Cholangitis, Sclerosing; Cholestasis, Extrahepatic; Cholic Acid; Choline Deficiency; Common Bile Duct; Disease Models, Animal; Epithelial Cells; Ethionine; Keratin-19; Ligation; Liver; Liver Cirrhosis, Biliary; Liver Regeneration; Male; Mice, Knockout; Phenotype; Pyridines; Signal Transduction; Stem Cells; Time Factors | 2015 |