Page last updated: 2024-08-24

di-n-propyldithiocarbamate and ditiocarb

di-n-propyldithiocarbamate has been researched along with ditiocarb in 5 studies

*Ditiocarb: A chelating agent that has been used to mobilize toxic metals from the tissues of humans and experimental animals. It is the main metabolite of DISULFIRAM. [MeSH]

*Ditiocarb: A chelating agent that has been used to mobilize toxic metals from the tissues of humans and experimental animals. It is the main metabolite of DISULFIRAM. [MeSH]

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19901 (20.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (20.00)24.3611
2020's3 (60.00)2.80

Authors

AuthorsStudies
Jones, MM; Topping, RJ1
Fu, Y; Guo, R; Huang, T; Li, C; Li, X; Liu, Y; Wang, T; Yang, Y; Zhang, P; Zhang, Z; Zhou, P1
Feng, J; Feng, R; Hou, Q; Li, C; Li, Y; Wang, S; Xu, R; Zhang, L1
Chen, H; Feng, J; Guan, D; Li, C; Li, Y; Xu, Z; Zhai, X; Zhang, L1
Han, X; Huang, T; Li, C; Li, Y; Lou, L; Wang, H1

Other Studies

5 other study(ies) available for di-n-propyldithiocarbamate and ditiocarb

ArticleYear
Optimal dithiocarbamate structure for immunomodulator action.
    Medical hypotheses, 1988, Volume: 27, Issue:1

    Topics: Acquired Immunodeficiency Syndrome; Adjuvants, Immunologic; Animals; Ditiocarb; Half-Life; Humans; Hydrogen-Ion Concentration; Pyrrolidines; Structure-Activity Relationship; Thiocarbamates

1988
The novel dithiocarbamate, DpdtC suppresses HER2-overexpressed cancer cells by up-regulating NDRG1 via inactivation of HER2-ERK 1/2 signaling.
    Scientific reports, 2018, 02-21, Volume: 8, Issue:1

    Topics: Animals; Breast Neoplasms; Cell Cycle Proteins; Cell Line, Tumor; Cell Proliferation; Ditiocarb; Female; Gene Expression Regulation, Neoplastic; Humans; Intracellular Signaling Peptides and Proteins; MAP Kinase Signaling System; Mice; Mice, Inbred BALB C; Mice, Nude; Ovarian Neoplasms; Phosphorylation; Receptor, ErbB-2; Signal Transduction; Up-Regulation

2018
DpdtC-Induced EMT Inhibition in MGC-803 Cells Was Partly through Ferritinophagy-Mediated ROS/p53 Pathway.
    Oxidative medicine and cellular longevity, 2020, Volume: 2020

    Topics: Autophagy; Ditiocarb; Epithelial-Mesenchymal Transition; Ferritins; Humans; Reactive Oxygen Species; Tumor Suppressor Protein p53

2020
The vicious cycle between ferritinophagy and ROS production triggered EMT inhibition of gastric cancer cells was through p53/AKT/mTor pathway.
    Chemico-biological interactions, 2020, Sep-01, Volume: 328

    Topics: Autophagy; Cell Line, Tumor; Ditiocarb; Epithelial-Mesenchymal Transition; Ferritins; Humans; Proto-Oncogene Proteins c-akt; Reactive Oxygen Species; Signal Transduction; Stomach Neoplasms; TOR Serine-Threonine Kinases; Transforming Growth Factor beta1; Tumor Suppressor Protein p53

2020
The interaction between lipocalin 2 and dipyridine ketone hydrazone dithiocarbamte may influence respective function in proliferation and metastasis-related gene expressions in HepG2 cell.
    Journal of biological inorganic chemistry : JBIC : a publication of the Society of Biological Inorganic Chemistry, 2021, Volume: 26, Issue:1

    Topics: Antineoplastic Agents; Autophagy; Cell Proliferation; Ditiocarb; Ferritins; Gene Expression Regulation, Neoplastic; Hep G2 Cells; Humans; Hydrazones; Iron Chelating Agents; Lipid Peroxidation; Lipocalin-2; Reactive Oxygen Species; Thiocarbamates; Transient Receptor Potential Channels

2021