deferoxamine and aspartic acid

deferoxamine has been researched along with aspartic acid in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (25.00)18.2507
2000's3 (75.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bonner, MJ; Gilman, SC; Pellmar, TC1
de Groot, H; Rauen, U; Wille, T1
Amaniti, E; Kouvelas, D; Lallas, A; Papazisis, G; Pourzitaki, C; Sardeli, C1
Guo, Y; Li, B; Li, C; Sun, M; Yu, J; Zhang, Y1

Other Studies

4 other study(ies) available for deferoxamine and aspartic acid

ArticleYear
Free radicals enhance basal release of D-[3H]aspartate from cerebral cortical synaptosomes.
    Journal of neurochemistry, 1994, Volume: 62, Issue:5

    Topics: Animals; Aspartic Acid; Calcium; Cerebral Cortex; Deferoxamine; Free Radicals; Guinea Pigs; Hydrogen Peroxide; Iron; Male; Synaptosomes; Tritium

1994
Improvement of the cold storage of blood vessels with a vascular preservation solution. Study in porcine aortic segments.
    Journal of vascular surgery, 2008, Volume: 47, Issue:2

    Topics: Adenosine; Alanine; Allopurinol; Animals; Aorta; Aspartic Acid; Buffers; Cell Survival; Citrates; Cold Temperature; Deferoxamine; Endothelial Cells; Glucose; Glutathione; Glycine; Histidine; Hydrogen-Ion Concentration; Insulin; Iron; Iron Chelating Agents; Mannitol; Membrane Potential, Mitochondrial; Mitochondria; Organ Preservation; Organ Preservation Solutions; Platelet Adhesiveness; Potassium Chloride; Procaine; Raffinose; Rewarming; Swine; Time Factors

2008
Deferoxamine decreases the excitatory amino acid levels and improves the histological outcome in the hippocampus of neonatal rats after hypoxia-ischemia.
    Pharmacological research, 2008, Volume: 57, Issue:1

    Topics: Animals; Animals, Newborn; Aspartic Acid; Deferoxamine; Excitatory Amino Acids; Female; Glutamic Acid; Hippocampus; Hypoxia-Inducible Factor 1, alpha Subunit; Hypoxia-Ischemia, Brain; Male; Neuroprotective Agents; Oxidative Stress; Rats; Rats, Wistar

2008
Iron is a potential key mediator of glutamate excitotoxicity in spinal cord motor neurons.
    Brain research, 2009, Feb-27, Volume: 1257

    Topics: Analysis of Variance; Animals; Aspartic Acid; Cation Transport Proteins; Deferoxamine; Ferritins; Glutamic Acid; Immunoblotting; Immunohistochemistry; In Vitro Techniques; Iron; Iron Chelating Agents; L-Lactate Dehydrogenase; Lipid Peroxidation; Motor Neurons; Nerve Degeneration; Neurotoxins; Rats; Rats, Sprague-Dawley; Receptors, Transferrin; Spinal Cord

2009