declopramide has been researched along with procainamide in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (40.00) | 18.2507 |
2000's | 3 (60.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Hua, J; Pero, RW | 1 |
Hua, J; Kane, R; Pero, RW | 1 |
Ivars, F; Leanderson, T; Lindgren, H; Pero, RW | 1 |
Leanderson, T; Lindgren, H; Olsson, AR; Pero, RW | 2 |
5 other study(ies) available for declopramide and procainamide
Article | Year |
---|---|
Toxicity, antitumor and chemosensitizing effects of 3-chloroprocainamide.
Topics: Animals; Antineoplastic Agents; Astrocytoma; Brain Neoplasms; Cisplatin; Dose-Response Relationship, Drug; Drug Interactions; Drug Screening Assays, Antitumor; Female; Humans; Male; Metoclopramide; Mice; Mice, SCID; Neoplasm Transplantation; Procainamide; Transplantation, Heterologous | 1997 |
Pharmacokinetics and central nervous system toxicity of declopramide (3-chloroprocainamide) in rats and mice.
Topics: Administration, Oral; Animals; Antiemetics; Biological Availability; Central Nervous System; Dopamine Antagonists; Dose-Response Relationship, Drug; Female; Metoclopramide; Mice; Mice, SCID; Procainamide; Rats; Rats, Wistar; Receptors, Dopamine D2; Receptors, Serotonin; Receptors, Serotonin, 5-HT3; Sleep Stages; Tissue Distribution | 1999 |
N-substituted benzamides inhibit nuclear factor-kappaB and nuclear factor of activated T cells activity while inducing activator protein 1 activity in T lymphocytes.
Topics: Acecainide; Benzamides; CD40 Ligand; Cell Nucleus; DNA-Binding Proteins; Enzyme Activation; Humans; Interleukin-2; Jurkat Cells; NF-kappa B; NF-kappa B p50 Subunit; NFATC Transcription Factors; Nuclear Proteins; Procainamide; Promoter Regions, Genetic; T-Lymphocytes; Transcription Factor AP-1; Transcription Factors; Transcription, Genetic | 2001 |
Mechanism of action for N-substituted benzamide-induced apoptosis.
Topics: Apoptosis; Benzamides; Caspase 9; Caspases; Cytochrome c Group; Enzyme Activation; G2 Phase; HL-60 Cells; Humans; Metoclopramide; Mitosis; Procainamide; Proto-Oncogene Proteins c-bcl-2; Tumor Suppressor Protein p53 | 2002 |
Differential usage of IkappaBalpha and IkappaBbeta in regulation of apoptosis versus gene expression.
Topics: Animals; Apoptosis; B-Lymphocytes; Cell Line; Flow Cytometry; Gene Expression Regulation; I-kappa B Proteins; Immunoglobulins; Lipopolysaccharides; Mice; Molecular Structure; NF-kappa B; NF-KappaB Inhibitor alpha; Procainamide; Proto-Oncogene Proteins c-bcl-2 | 2003 |