dactinomycin and vitamin u

dactinomycin has been researched along with vitamin u in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	1 (50.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Nezu, JI; Ohashi, R; Oku, A; Sai, Y; Shimane, M; Tamai, I; Tsuji, A; Yabuuchi, H	1
Depatie, C; Hayakawa, J; Mercola, D; Ohmichi, M	1

Other Studies

2 other study(ies) available for dactinomycin and vitamin u

Article	Year
Na(+)-dependent carnitine transport by organic cation transporter (OCTN2): its pharmacological and toxicological relevance. The Journal of pharmacology and experimental therapeutics, 1999, Volume: 291, Issue:2 Topics: Biological Transport, Active; Carnitine; Carrier Proteins; Cations; Cells, Cultured; Dose-Response Relationship, Drug; Embryo, Mammalian; Humans; Hydrogen-Ion Concentration; Kidney; Membrane Proteins; Organic Cation Transport Proteins; Sodium; Solute Carrier Family 22 Member 5; Stereoisomerism	1999
The activation of c-Jun NH2-terminal kinase (JNK) by DNA-damaging agents serves to promote drug resistance via activating transcription factor 2 (ATF2)-dependent enhanced DNA repair. The Journal of biological chemistry, 2003, Jun-06, Volume: 278, Issue:23 Topics: Activating Transcription Factor 2; Antibiotics, Antineoplastic; Antineoplastic Agents; Antineoplastic Agents, Phytogenic; Breast Neoplasms; Cisplatin; Cyclic AMP Response Element-Binding Protein; Dactinomycin; DNA Damage; DNA Repair; Drug Resistance, Neoplasm; Enzyme Inhibitors; Etoposide; Gastrointestinal Agents; Humans; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinases; Phenotype; Transcription Factors; Transcriptional Activation; Tumor Cells, Cultured; Vitamin U	2003

Article

Year

Na(+)-dependent carnitine transport by organic cation transporter (OCTN2): its pharmacological and toxicological relevance.

The Journal of pharmacology and experimental therapeutics, 1999, Volume: 291, Issue:2

Topics: Biological Transport, Active; Carnitine; Carrier Proteins; Cations; Cells, Cultured; Dose-Response Relationship, Drug; Embryo, Mammalian; Humans; Hydrogen-Ion Concentration; Kidney; Membrane Proteins; Organic Cation Transport Proteins; Sodium; Solute Carrier Family 22 Member 5; Stereoisomerism

1999

The activation of c-Jun NH2-terminal kinase (JNK) by DNA-damaging agents serves to promote drug resistance via activating transcription factor 2 (ATF2)-dependent enhanced DNA repair.

The Journal of biological chemistry, 2003, Jun-06, Volume: 278, Issue:23

Topics: Activating Transcription Factor 2; Antibiotics, Antineoplastic; Antineoplastic Agents; Antineoplastic Agents, Phytogenic; Breast Neoplasms; Cisplatin; Cyclic AMP Response Element-Binding Protein; Dactinomycin; DNA Damage; DNA Repair; Drug Resistance, Neoplasm; Enzyme Inhibitors; Etoposide; Gastrointestinal Agents; Humans; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinases; Phenotype; Transcription Factors; Transcriptional Activation; Tumor Cells, Cultured; Vitamin U

2003