cuprizone has been researched along with myelin oligodendrocyte glycoprotein (35-55) in 9 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (11.11) | 29.6817 |
| 2010's | 8 (88.89) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Correcha, M; Fordham, SA; Liñares, D; Maña, P; Silva, D; Staykova, MA; Willenborg, DO | 1 |
| Choi, K; Darnall, L; Hu, T; Lane, TE; Liu, L; Ransohoff, RM | 1 |
| Bauer, J; Binder, CJ; Bradl, M; Haider, L; Hametner, S; Lassmann, H; Liblau, RS; Mahad, D; Probert, L; Schuh, C; Smith, KJ; Van Dam, AM; Wimmer, I | 1 |
| Baxi, EG; Calabresi, PA; DeBruin, J; Fairchild, AN; Gocke, AR; Grishkan, IV; Kirby, LA; Smith, MD; Strasburger, HJ; Tosi, DM | 1 |
| Baxi, EG; Glenn, JD; Kirby, LA; Smith, MD; Whartenby, KA | 1 |
| Beyer, C; Clarner, T; Dreymüller, D; Fallier-Becker, P; Große-Veldmann, R; Kipp, M; Ohl, K; Rüther, BJ; Scheld, M; Tenbrock, K; Zendedel, A | 1 |
| Abbracchio, MP; Coppolino, GT; Dimou, L; Fumagalli, M; Furlan, R; Gelosa, P; Lecca, D; Marangon, D; Menichetti, G; Negri, C | 1 |
| Amor, S; Beyer, C; Bhattarai, S; Chrzanowski, U; Clarner, T; Denecke, B; Fallier-Becker, P; Hochstrasser, T; Horn-Bochtler, A; Kipp, M; Nyamoya, S; Rohr, SO; Scheld, M; Schmitz, C; Schweiger, F | 1 |
| Behrangi, N; Hochstrasser, T; Horn, A; Kipp, M; Schmitz, C; Schweiger, F; Yakimov, V; Zhan, J | 1 |
9 other study(ies) available for cuprizone and myelin oligodendrocyte glycoprotein (35-55)
| Article | Year |
|---|---|
Demyelination caused by the copper chelator cuprizone halts T cell mediated autoimmune neuroinflammation.
Topics: Animals; Antibody Specificity; Antigens; Chelating Agents; Cuprizone; Cytokines; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Female; Glycoproteins; Immune Tolerance; Lymphocyte Activation; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Myelin Proteins; Myelin Sheath; Myelin-Oligodendrocyte Glycoprotein; Peptide Fragments; T-Lymphocytes | 2009 |
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
Topics: Animals; Animals, Newborn; Antibodies; Bone Marrow; Cell Differentiation; Central Nervous System Stimulants; Cerebellum; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Flow Cytometry; Freund's Adjuvant; Glycoproteins; In Vitro Techniques; Leukocyte Common Antigens; Lipopolysaccharides; Mice; Mice, Inbred C57BL; Mice, Knockout; Microscopy, Electron, Transmission; Myelin Basic Protein; Myelin Proteolipid Protein; Myelin Sheath; Myelin-Oligodendrocyte Glycoprotein; Nerve Regeneration; Neurologic Examination; Oligodendroglia; Peptide Fragments; Picrotoxin; Proliferating Cell Nuclear Antigen; Receptors, Interleukin-8B; Recovery of Function; Severity of Illness Index; Stem Cells; Time Factors | 2010 |
Oxidative tissue injury in multiple sclerosis is only partly reflected in experimental disease models.
Topics: Aging; Animals; CD4 Antigens; CD8 Antigens; Coronavirus Infections; Cuprizone; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Gene Expression; Iron; Lipopolysaccharides; Macrophages; Mice, Inbred C57BL; Microglia; Multiple Sclerosis; Myelin-Oligodendrocyte Glycoprotein; Oxidative Stress; Peptide Fragments; Rats; Rats, Inbred Lew; Respiratory Burst; T-Lymphocytes | 2014 |
Transfer of myelin-reactive th17 cells impairs endogenous remyelination in the central nervous system of cuprizone-fed mice.
Topics: Adoptive Transfer; Animals; Cell Polarity; Cells, Cultured; Central Nervous System; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Freund's Adjuvant; Interleukin-17; Leukocyte Common Antigens; Mice; Mice, Inbred C57BL; Mice, Transgenic; Monoamine Oxidase Inhibitors; Monocytes; Myelin Proteins; Myelin Sheath; Myelin-Oligodendrocyte Glycoprotein; Neutrophil Infiltration; Peptide Fragments; Regeneration; Th17 Cells; Time Factors | 2015 |
Disparate Effects of Mesenchymal Stem Cells in Experimental Autoimmune Encephalomyelitis and Cuprizone-Induced Demyelination.
Topics: Animals; CD4-Positive T-Lymphocytes; Cell Death; Cell Proliferation; Corpus Callosum; Cuprizone; Cytokines; Demyelinating Diseases; Encephalomyelitis, Autoimmune, Experimental; Female; Lymphocyte Subsets; Mesenchymal Stem Cell Transplantation; Mesenchymal Stem Cells; Mice, Inbred C57BL; Myelin-Oligodendrocyte Glycoprotein; Oligodendroglia; Peptide Fragments | 2015 |
Neurodegeneration Triggers Peripheral Immune Cell Recruitment into the Forebrain.
Topics: Adoptive Transfer; Animals; Calcium-Binding Proteins; CD3 Complex; Chelating Agents; Cuprizone; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Female; Freund's Adjuvant; Lymph Nodes; Lymphocytes; Mice; Mice, Inbred C57BL; Microfilament Proteins; Monocytes; Myelin-Oligodendrocyte Glycoprotein; Neurodegenerative Diseases; Peptide Fragments; Pertussis Toxin; Prosencephalon | 2016 |
Differential local tissue permissiveness influences the final fate of GPR17-expressing oligodendrocyte precursors in two distinct models of demyelination.
Topics: Animals; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Green Fluorescent Proteins; Male; Mice, Inbred C57BL; Mice, Transgenic; Myelin-Oligodendrocyte Glycoprotein; Nerve Tissue Proteins; Oligodendrocyte Precursor Cells; Peptide Fragments; Receptors, G-Protein-Coupled; Remyelination; Spinal Cord | 2018 |
Oligodendrocyte degeneration and concomitant microglia activation directs peripheral immune cells into the forebrain.
Topics: Animals; Cuprizone; Encephalomyelitis, Autoimmune, Experimental; Female; Immunity, Cellular; Male; Mice; Mice, Inbred C57BL; Microglia; Multiple Sclerosis; Myelin-Oligodendrocyte Glycoprotein; Oligodendroglia; Peptide Fragments; Prosencephalon | 2019 |
Continuous cuprizone intoxication allows active experimental autoimmune encephalomyelitis induction in C57BL/6 mice.
Topics: Administration, Oral; Animals; Apoptosis; Cuprizone; Encephalomyelitis, Autoimmune, Experimental; Female; Mice; Mice, Inbred C57BL; Myelin-Oligodendrocyte Glycoprotein; Oligodendroglia; Peptide Fragments | 2019 |