cuprizone has been researched along with lysophosphatidylcholines in 16 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (6.25) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (18.75) | 29.6817 |
2010's | 10 (62.50) | 24.3611 |
2020's | 2 (12.50) | 2.80 |
Authors | Studies |
---|---|
Blakemore, WF | 1 |
Franklin, RJ; Fruttiger, M; Richardson, WD; Woodruff, RH | 1 |
Alberta, JA; Arnett, HA; Fancy, SP; Franklin, RJ; Kaing, S; Plant, SR; Raine, CS; Rowitch, DH; Stiles, CD; Zhao, C | 1 |
Bernard, F; Chédotal, A; Franklin, RJ; Hu, B; Ji, B; Jung, V; Lee, X; Mason, J; Mi, S; Miklasz, S; Miller, RH; Pepinsky, B; Roulois, A; Shao, Z; Shea, D; Shields, CB; Tang, W; Wu, W; Xu, J; Zhang, Y | 1 |
Liu, B; Liu, Y; Sun, X; Xiao, Z; Zhang, L | 1 |
Rawji, KS; Yong, VW | 1 |
Bansal, R; Franklin, RJ; Furusho, M; Roulois, AJ | 1 |
Bittner, S; Budde, T; Cerina, M; Daldrup, T; Ehling, P; Göbel, K; Gudi, V; Herrmann, AM; Kleinschnitz, C; Meuth, P; Meuth, SG; Narayanan, V; Pape, HC; Ruck, T; Seidenbecher, T; Skripuletz, T; Stangel, M; Wiendl, H | 1 |
Brickey, WJ; David, CN; Freeman, L; Guo, H; Jha, S; Ting, JP | 1 |
Bahn, E; Brück, W; Junker, A; Scheidt, U; Schultz, V; Stadelmann, C; van der Meer, F; Wrzos, C | 1 |
Allinquant, B; Camand, E; Carrete, A; Chierto, E; Hendriks, JJA; Jafarian-Tehrani, M; Llufriu-Dabén, G; Mailleux, J; Massaad, C; Meffre, D; Rose, C; Simon, A; Vanmierlo, T | 1 |
Budde, T; Cerina, M; Daldrup, T; Fernandez-Orth, J; Göbel, K; Gudi, V; Herrmann, AM; Lesting, J; Meuth, P; Meuth, SG; Narayanan, V; Pape, HC; Schiffler, P; Seidenbecher, T; Skripuletz, T; Stangel, M; Wiendl, H | 1 |
Baumgartner, L; de Vries, R; Galeno, G; Good, A; Good, N; Hlavica, M; Hooijmans, CR; Ineichen, BV; Jung, T; Schneider, MP; Schuler, FAF | 1 |
Franklin, RJM; McMurran, CE; Zhao, C | 1 |
Choi, CI; Kim, HN; Kleppe, LS; Langley, MR; Scarisbrick, IA; Simon, WL; Triplet, EM; Yoon, H | 1 |
Huang, JK; Kouki, T; Ohno, N; Osanai, Y; Yamazaki, R | 1 |
2 review(s) available for cuprizone and lysophosphatidylcholines
Article | Year |
---|---|
The benefits and detriments of macrophages/microglia in models of multiple sclerosis.
Topics: Animals; Cell Movement; Central Nervous System; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Humans; Immunologic Factors; Lysophosphatidylcholines; Macrophages; Microglia; Multiple Sclerosis; Neurons; Oligodendroglia; T-Lymphocytes | 2013 |
Remyelination promoting therapies in multiple sclerosis animal models: a systematic review and meta-analysis.
Topics: Animals; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Ethidium; Eye Proteins; Lysophosphatidylcholines; Mice; Multiple Sclerosis; Myelin Sheath; Nerve Growth Factors; Oligodendrocyte Precursor Cells; Oligodendroglia; Platelet-Derived Growth Factor; Remyelination; Serpins; Tocopherols | 2019 |
14 other study(ies) available for cuprizone and lysophosphatidylcholines
Article | Year |
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Remyelination in the CNS.
Topics: 6-Aminonicotinamide; Animals; Axons; Brain; Cats; Cuprizone; Lysophosphatidylcholines; Myelin Sheath; Oligodendroglia; Rabbits; Spinal Cord | 1981 |
Platelet-derived growth factor regulates oligodendrocyte progenitor numbers in adult CNS and their response following CNS demyelination.
Topics: Animals; Central Nervous System; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Glial Fibrillary Acidic Protein; Lysophosphatidylcholines; Mice; Mice, Transgenic; Nerve Regeneration; Oligodendroglia; Platelet-Derived Growth Factor; Promoter Regions, Genetic; Stem Cells; Up-Regulation | 2004 |
bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.
Topics: Animals; Animals, Newborn; Basic Helix-Loop-Helix Transcription Factors; Brain; Cell Nucleus; Cuprizone; Cytoplasm; Demyelinating Diseases; DNA-Binding Proteins; Ethidium; Humans; Lysophosphatidylcholines; Mice; Mice, Inbred C57BL; Multiple Sclerosis; Myelin Sheath; Nerve Tissue Proteins; Oligodendrocyte Transcription Factor 2; Oligodendroglia; Rats; Rats, Sprague-Dawley; Spinal Cord; Stem Cells; Transcription Factors | 2004 |
Promotion of central nervous system remyelination by induced differentiation of oligodendrocyte precursor cells.
Topics: Animals; Animals, Newborn; Antibodies; Cell Differentiation; Cells, Cultured; Cuprizone; Demyelinating Autoimmune Diseases, CNS; Disease Models, Animal; Ganglia, Spinal; Lysophosphatidylcholines; Membrane Proteins; Mice; Myelin Proteins; Myelin Sheath; Nerve Tissue Proteins; Oligodendroglia; Organ Culture Techniques; Rats; Rats, Sprague-Dawley; Stem Cells | 2009 |
Rolipram promotes remyelination possibly via MEK-ERK signal pathway in cuprizone-induced demyelination mouse.
Topics: Animals; Blotting, Western; Brain; Cell Differentiation; Cuprizone; Demyelinating Diseases; Lysophosphatidylcholines; MAP Kinase Signaling System; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Oligodendroglia; Organ Culture Techniques; Phosphodiesterase 4 Inhibitors; Rats; Rats, Sprague-Dawley; Rolipram; Stem Cells | 2012 |
Fibroblast growth factor signaling in oligodendrocyte-lineage cells facilitates recovery of chronically demyelinated lesions but is redundant in acute lesions.
Topics: 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase; Animals; Animals, Newborn; Basic Helix-Loop-Helix Transcription Factors; Cell Differentiation; Cell Lineage; Cells, Cultured; Chelating Agents; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Fibroblast Growth Factors; Lysophosphatidylcholines; Mice; Mice, Inbred C57BL; Mice, Transgenic; Oligodendroglia; Receptor, Fibroblast Growth Factor, Type 1; Receptor, Fibroblast Growth Factor, Type 2; Recovery of Function; Signal Transduction; Spinal Cord | 2015 |
The quality of cortical network function recovery depends on localization and degree of axonal demyelination.
Topics: Adaptive Immunity; Animals; Behavior, Animal; Cerebral Cortex; Cuprizone; Demyelinating Diseases; Electrodes, Implanted; Gray Matter; Lysophosphatidylcholines; Mice; Mice, Inbred C57BL; Myelin Sheath; Nerve Net; Recovery of Function; Sensation; White Matter | 2017 |
NLR members NLRC4 and NLRP3 mediate sterile inflammasome activation in microglia and astrocytes.
Topics: Animals; Apoptosis Regulatory Proteins; Astrocytes; Calcium-Binding Proteins; Cuprizone; Disease Models, Animal; Inflammasomes; Inflammation; Lysophosphatidylcholines; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Microglia; NLR Family, Pyrin Domain-Containing 3 Protein | 2017 |
Acutely damaged axons are remyelinated in multiple sclerosis and experimental models of demyelination.
Topics: Amyloid beta-Protein Precursor; Animals; Axons; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Glial Fibrillary Acidic Protein; Lysophosphatidylcholines; Male; Membrane Proteins; Mice; Mice, Inbred C57BL; Microglia; Monoamine Oxidase Inhibitors; Multiple Sclerosis; Myelin Sheath; Rats; Rats, Inbred Lew; Statistics, Nonparametric; Time Factors | 2017 |
Targeting demyelination via α-secretases promoting sAPPα release to enhance remyelination in central nervous system.
Topics: Amyloid beta-Protein Precursor; Amyloid Precursor Protein Secretases; Animals; Axons; Brain; Cells, Cultured; Cerebellum; Corpus Callosum; Cuprizone; Demyelinating Diseases; Etazolate; Lysophosphatidylcholines; Male; Mice, Inbred C57BL; Myelin Sheath; Neuroprotective Agents; Remyelination | 2018 |
Impairment of frequency-specific responses associated with altered electrical activity patterns in auditory thalamus following focal and general demyelination.
Topics: Acoustic Stimulation; Action Potentials; Animals; Auditory Cortex; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Functional Laterality; Geniculate Bodies; Gliosis; Gray Matter; Lysophosphatidylcholines; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Myelin Proteolipid Protein; Neurons; Psychoacoustics; Thalamus | 2018 |
Toxin-Based Models to Investigate Demyelination and Remyelination.
Topics: Animals; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Ethidium; Lysophosphatidylcholines; Mice; Mice, Inbred C57BL; Rats; Remyelination | 2019 |
Blocking the Thrombin Receptor Promotes Repair of Demyelinated Lesions in the Adult Brain.
Topics: Animals; Astrocytes; Axons; Brain-Derived Neurotrophic Factor; Chelating Agents; Coculture Techniques; Copper; Corpus Callosum; Cuprizone; Demyelinating Diseases; Gene Expression Profiling; Lysophosphatidylcholines; Male; Mice; Mice, Knockout; Myelin Sheath; Nerve Regeneration; Neural Stem Cells; Oligodendroglia; Receptor, PAR-1; Rotarod Performance Test; Spinal Cord; White Matter | 2020 |
Pharmacological treatment promoting remyelination enhances motor function after internal capsule demyelination in mice.
Topics: Animals; Clemastine; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Internal Capsule; Lysophosphatidylcholines; Mice; Mice, Inbred C57BL; Multiple Sclerosis; Myelin Sheath; Oligodendroglia; Remyelination | 2023 |