Page last updated: 2024-09-05

crambescidin 816 and crambescidin 800

crambescidin 816 has been researched along with crambescidin 800 in 3 studies

Compound Research Comparison

Studies
(crambescidin 816)
Trials
(crambescidin 816)
Recent Studies (post-2010)
(crambescidin 816)
Studies
(crambescidin 800)
Trials
(crambescidin 800)
Recent Studies (post-2010) (crambescidin 800)
7051206

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Overman, LE; Rhee, YH1
Botana, LM; López-Alonso, H; Rubiolo, JA; Ternon, E; Thomas, OP; Vega, FV; Vieytes, MR1
Botana, LM; Cabezas-Sainz, P; Guerra-Varela, J; López, R; Roel, M; Rubiolo, JA; Sánchez, L; Silva, SB; Thomas, OP1

Other Studies

3 other study(ies) available for crambescidin 816 and crambescidin 800

ArticleYear
Total synthesis of (-)-crambidine and definition of the relative configuration of its unique tetracyclic guanidinium core.
    Journal of the American Chemical Society, 2005, Nov-09, Volume: 127, Issue:44

    Topics: Alkaloids; Guanidine; Spiro Compounds; Stereoisomerism

2005
Crambescidin-816 acts as a fungicidal with more potency than crambescidin-800 and -830, inducing cell cycle arrest, increased cell size and apoptosis in Saccharomyces cerevisiae.
    Marine drugs, 2013, Nov-08, Volume: 11, Issue:11

    Topics: Alkaloids; Apoptosis; Cell Cycle Checkpoints; Cell Death; Cell Division; Cell Size; Fungicides, Industrial; G2 Phase; Guanidine; Membrane Potential, Mitochondrial; Saccharomyces cerevisiae; Spiro Compounds

2013
Marine guanidine alkaloids crambescidins inhibit tumor growth and activate intrinsic apoptotic signaling inducing tumor regression in a colorectal carcinoma zebrafish xenograft model.
    Oncotarget, 2016, Dec-13, Volume: 7, Issue:50

    Topics: Alkaloids; Animals; Antineoplastic Agents; Apoptosis; Caspase 3; Cell Adhesion; Cell Cycle Proteins; Cell Proliferation; Cell Survival; Colorectal Neoplasms; Cytoskeleton; Dose-Response Relationship, Drug; G1 Phase Cell Cycle Checkpoints; Guanidine; Hep G2 Cells; HT29 Cells; Humans; Inhibitory Concentration 50; MCF-7 Cells; Membrane Potential, Mitochondrial; Signal Transduction; Spiro Compounds; Time Factors; Tumor Burden; Xenograft Model Antitumor Assays; Zebrafish

2016