cocaine and kn 62

cocaine has been researched along with kn 62 in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's1 (33.33)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Krnjević, K; Liu, PL; McArdle, JJ; Ren, J; Ye, JH1
Cuevas, J; Henning, RJ1
Abbott, TB; Chung, L; Colangelo, CM; Gulcicek, EE; Lam, TT; Nairn, AC; Rich, MT; Stone, KL; Taylor, JR; Torregrossa, MM1

Other Studies

3 other study(ies) available for cocaine and kn 62

ArticleYear
Cocaine decreases the glycine-induced Cl- current of acutely dissociated rat hippocampal neurons.
    European journal of pharmacology, 1999, Feb-12, Volume: 367, Issue:1

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; Alkaloids; Anesthetics, Local; Animals; Benzophenanthridines; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Chloride Channels; Cocaine; Cyclic AMP-Dependent Protein Kinases; Dose-Response Relationship, Drug; Drug Interactions; Electric Stimulation; Enzyme Inhibitors; Excitatory Amino Acid Agonists; Glycine; Glycine Agents; Hippocampus; Isoquinolines; Kainic Acid; Membrane Potentials; Neurons; Patch-Clamp Techniques; Phenanthridines; Protein Kinase C; Rats; Rats, Sprague-Dawley; Strychnine; Sulfonamides; Tetrodotoxin

1999
Cocaine activates calcium/calmodulin kinase II and causes cardiomyocyte hypertrophy.
    Journal of cardiovascular pharmacology, 2006, Volume: 48, Issue:1

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; Adenosine Triphosphate; Animals; Calcium Channels; Calcium Signaling; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Cells, Cultured; Cocaine; Dose-Response Relationship, Drug; Heart Ventricles; Hypertrophy; Male; Membrane Potentials; Myocytes, Cardiac; Myosin Heavy Chains; Patch-Clamp Techniques; Peptides; Phenylephrine; Phosphorus Radioisotopes; Phosphorylation; Rats; Rats, Sprague-Dawley; Sarcolemma

2006
Phosphoproteomic Analysis Reveals a Novel Mechanism of CaMKIIα Regulation Inversely Induced by Cocaine Memory Extinction versus Reconsolidation.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2016, 07-20, Volume: 36, Issue:29

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; Amygdala; Animals; Benzylamines; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Cocaine; Conditioning, Operant; Cues; Enzyme Inhibitors; Extinction, Psychological; HEK293 Cells; Humans; Male; Memory; Phosphorylation; Proteomics; Rats; Rats, Sprague-Dawley; Self Administration; Serine; Signal Transduction; Sulfonamides

2016