cholic acid has been researched along with diethylnitrosamine in 5 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (40.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (40.00) | 24.3611 |
| 2020's | 1 (20.00) | 2.80 |
| Authors | Studies |
|---|---|
| Delzenne, NM; Roberfroid, MB; Taper, HS | 1 |
| Ito, H; Kawasaki, H; Noguchi, N; Oyama, K; Shiota, G; Takano, T | 1 |
| Buckley, K; Guo, GL; Kong, B; Li, G; Luyendyk, JP; Tawfik, O; Williams, JA; Zhu, Y | 1 |
| Apte, U; Beggs, K; Bhushan, B; Borude, P; Edwards, G; Gunewardena, S; Li, H; Manley, MW; O'Neil, M; Roy, N; Sun, L; Walesky, C | 1 |
| Chun, HJ; Kwon, YH; Shim, YJ | 1 |
5 other study(ies) available for cholic acid and diethylnitrosamine
| Article | Year |
|---|---|
Changes in bile acids metabolism during rat hepatocarcinogenesis: causative or unrelated?
Topics: 2-Acetylaminofluorene; Animals; Bile Acids and Salts; Cholic Acid; Cholic Acids; Deoxycholic Acid; Diethylnitrosamine; Lithocholic Acid; Liver; Liver Neoplasms, Experimental; Male; Rats; Rats, Wistar | 1994 |
Oral administration of cholic acid promotes growth of liver tumors initiated by diethylnitrosamine in rats.
Topics: Administration, Oral; Animals; Apoptosis; Carcinogens; Cell Division; Cholic Acid; Diethylnitrosamine; Disease Progression; Immunohistochemistry; In Situ Nick-End Labeling; Liver Neoplasms, Experimental; Male; Rats; Rats, Inbred F344 | 1999 |
Mice with hepatocyte-specific FXR deficiency are resistant to spontaneous but susceptible to cholic acid-induced hepatocarcinogenesis.
Topics: Animals; Apoptosis; Carcinogenesis; Cell Proliferation; Cholic Acid; Cyclin D1; Diethylnitrosamine; Hepatocytes; Janus Kinases; Liver Neoplasms; Mice; Mice, Knockout; Mitogen-Activated Protein Kinase Kinases; Models, Animal; Receptors, Cytoplasmic and Nuclear; Signal Transduction; STAT3 Transcription Factor; Tumor Suppressor Protein p53 | 2016 |
Bile acids promote diethylnitrosamine-induced hepatocellular carcinoma via increased inflammatory signaling.
Topics: Adult; Animals; Apoptosis; Carcinoma, Hepatocellular; Cell Proliferation; Cell Transformation, Neoplastic; Cells, Cultured; Cholic Acid; Diethylnitrosamine; Endoplasmic Reticulum Stress; Female; Gene Expression Regulation, Neoplastic; Hepatocytes; Humans; Inflammation Mediators; Kruppel-Like Factor 4; Liver Neoplasms, Experimental; Male; Mice, Inbred C57BL; Middle Aged; Mitogen-Activated Protein Kinases; Neoplastic Stem Cells; NF-kappa B; Signal Transduction; Tumor Burden; Young Adult | 2016 |
Cholic acid supplementation accelerates the progression of nonalcoholic fatty liver disease to the procarcinogenic state in mice fed a high-fat and high-cholesterol diet.
Topics: Amino Acids; Animals; Carcinogenesis; Carcinoma, Hepatocellular; Cholesterol; Cholesterol, Dietary; Cholic Acid; Diet, High-Fat; Dietary Supplements; Diethylnitrosamine; Disease Progression; Gene Expression Regulation; Lipid Metabolism; Liver; Male; Metabolic Networks and Pathways; Mice; Mice, Inbred C57BL; Mitochondria, Liver; Non-alcoholic Fatty Liver Disease; Transcriptome | 2022 |