celecoxib and isomethyleugenol

celecoxib has been researched along with isomethyleugenol in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (25.00)29.6817
2010's2 (50.00)24.3611
2020's1 (25.00)2.80

Authors

AuthorsStudies
Chuang, HC; Gaffney, KJ; Kardosh, A; Petasis, NA; Schönthal, AH1
Blechschmidt, C; Czabanka, M; Koch, A; Misch, M; Plotkin, M; Stockhammer, F; Tuettenberg, J; Vajkoczy, P1
Deckmann, K; Geisslinger, G; Grösch, S; Rörsch, F1
Kleszcz, R; Krajka-Kuźniak, V; Kruhlenia, N; Majchrzak-Celińska, A; Misiorek, JO; Przybyl, L; Rolle, K1

Other Studies

4 other study(ies) available for celecoxib and isomethyleugenol

ArticleYear
COX-2 inhibition is neither necessary nor sufficient for celecoxib to suppress tumor cell proliferation and focus formation in vitro.
    Molecular cancer, 2008, May-16, Volume: 7

    Topics: Celecoxib; Cell Line, Tumor; Cell Proliferation; Cell Survival; Cyclooxygenase 2; Cyclooxygenase Inhibitors; Humans; Immunoblotting; Methylation; Neoplasms; Pyrazoles; Sulfonamides; Transcription Factor CHOP

2008
Continuous low-dose temozolomide and celecoxib in recurrent glioblastoma.
    Journal of neuro-oncology, 2010, Volume: 100, Issue:3

    Topics: Adult; Aged; Antineoplastic Agents, Alkylating; Antineoplastic Combined Chemotherapy Protocols; Brain Neoplasms; Celecoxib; Cyclooxygenase 2 Inhibitors; Dacarbazine; Disease-Free Survival; DNA Modification Methylases; DNA Repair Enzymes; Female; Fluorine Radioisotopes; Follow-Up Studies; Glioblastoma; Humans; Male; Methylation; Middle Aged; Positron-Emission Tomography; Pyrazoles; Retrospective Studies; Sulfonamides; Temozolomide; Tumor Suppressor Proteins; Tyrosine

2010
Dimethylcelecoxib induces an inhibitory complex consisting of HDAC1/NF-κB(p65)RelA leading to transcriptional downregulation of mPGES-1 and EGR1.
    Cellular signalling, 2012, Volume: 24, Issue:2

    Topics: Celecoxib; Dinoprostone; Early Growth Response Protein 1; Electrophoretic Mobility Shift Assay; Female; HeLa Cells; Histone Deacetylase 1; Histone Deacetylase Inhibitors; Humans; Hydroxamic Acids; Immunoprecipitation; Intramolecular Oxidoreductases; Methylation; Promoter Regions, Genetic; Prostaglandin-E Synthases; Protein Binding; Pyrazoles; RNA, Small Interfering; Signal Transduction; Sulfonamides; Transcription Factor RelA; Transcription, Genetic; Transcriptional Activation; Uterine Cervical Neoplasms

2012
COXIBs and 2,5-dimethylcelecoxib counteract the hyperactivated Wnt/β-catenin pathway and COX-2/PGE2/EP4 signaling in glioblastoma cells.
    BMC cancer, 2021, May-03, Volume: 21, Issue:1

    Topics: Aged; Antineoplastic Agents, Alkylating; Apoptosis; beta Catenin; Brain Neoplasms; Celecoxib; Cell Cycle; Cell Line, Tumor; Cyclooxygenase 2; Cyclooxygenase 2 Inhibitors; Dinoprostone; DNA Modification Methylases; DNA Repair Enzymes; Dose-Response Relationship, Drug; Etoricoxib; Female; Glioblastoma; Humans; Isoxazoles; Lactones; Male; Methylation; Middle Aged; Neoplasm Proteins; Pyrazoles; Receptors, Prostaglandin E, EP4 Subtype; Sulfonamides; Sulfones; Temozolomide; Tumor Suppressor Proteins; Wnt Signaling Pathway

2021