catechin and involucrin

catechin has been researched along with involucrin in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's4 (80.00)29.6817
2010's1 (20.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Balasubramanian, S; Eckert, RL; Efimova, T1
Balasubramanian, S; Eckert, RL1
Balasubramanian, S; Crish, JF; Eckert, RL; Efimova, T2
Adhikary, G; Chew, YC; Eckert, RL; Reece, EA1

Reviews

1 review(s) available for catechin and involucrin

ArticleYear
Opposing action of curcumin and green tea polyphenol in human keratinocytes.
    Molecular nutrition & food research, 2006, Volume: 50, Issue:2

    Topics: Antioxidants; Catechin; Cell Differentiation; Chemoprevention; Curcumin; Flavonoids; Gene Expression; Humans; Keratinocytes; p38 Mitogen-Activated Protein Kinases; Phenols; Polyphenols; Protein Precursors; Signal Transduction; Tea

2006

Other Studies

4 other study(ies) available for catechin and involucrin

ArticleYear
Green tea polyphenol stimulates a Ras, MEKK1, MEK3, and p38 cascade to increase activator protein 1 factor-dependent involucrin gene expression in normal human keratinocytes.
    The Journal of biological chemistry, 2002, Jan-18, Volume: 277, Issue:3

    Topics: Catechin; Cells, Cultured; Gene Expression Regulation; Humans; Keratinocytes; MAP Kinase Kinase 3; MAP Kinase Kinase Kinase 1; MAP Kinase Signaling System; Mitogen-Activated Protein Kinase Kinases; Protein Precursors; Protein Serine-Threonine Kinases; Protein-Tyrosine Kinases; ras Proteins; Tea; Transcription Factor AP-1

2002
Green tea polyphenol and curcumin inversely regulate human involucrin promoter activity via opposing effects on CCAAT/enhancer-binding protein function.
    The Journal of biological chemistry, 2004, Jun-04, Volume: 279, Issue:23

    Topics: Antioxidants; Binding Sites; Catechin; CCAAT-Enhancer-Binding Proteins; Cell Differentiation; Curcumin; Cysteine Endopeptidases; DNA; Enzyme Activation; Flavonoids; Humans; Keratinocytes; MAP Kinase Kinase 3; MAP Kinase Kinase Kinase 1; MAP Kinase Kinase Kinases; Mitogen-Activated Protein Kinase 13; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinases; Multienzyme Complexes; Mutation; Phenols; Polyphenols; Promoter Regions, Genetic; Proteasome Endopeptidase Complex; Protein Precursors; Protein-Tyrosine Kinases; Tea; Transcription Factor CHOP; Transcription Factors

2004
Antioxidants regulate normal human keratinocyte differentiation.
    Biochemical pharmacology, 2004, Sep-15, Volume: 68, Issue:6

    Topics: Anticarcinogenic Agents; Catechin; CCAAT-Enhancer-Binding Proteins; Cell Differentiation; Curcumin; Drug Interactions; Humans; Keratinocytes; Mitogen-Activated Protein Kinase 13; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Promoter Regions, Genetic; Protein Precursors; Tea

2004
PKC-delta and -eta, MEKK-1, MEK-6, MEK-3, and p38-delta are essential mediators of the response of normal human epidermal keratinocytes to differentiating agents.
    The Journal of investigative dermatology, 2010, Volume: 130, Issue:8

    Topics: Anticarcinogenic Agents; Calcium; Carcinogens; Catechin; Cell Differentiation; Cells, Cultured; Epidermal Cells; Humans; Keratinocytes; MAP Kinase Kinase 3; MAP Kinase Kinase 6; MAP Kinase Kinase Kinase 1; MAP Kinase Signaling System; Mitogen-Activated Protein Kinase 13; Promoter Regions, Genetic; Protein Kinase C; Protein Kinase C-delta; Protein Precursors; RNA, Small Interfering; Tetradecanoylphorbol Acetate; Transcription Factor AP-1

2010