carbamates and hes1 protein, human

carbamates has been researched along with hes1 protein, human in 7 studies

Research

Studies (7)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's4 (57.14)29.6817
2010's3 (42.86)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Abdulkadir, SA; McDonald, JM; Zayzafoon, M1
Chung, IY; Kang, JH; Kim, HJ; Lee, DH; Lee, JS; Lee, YH; Lee, YS; Park, CS; Shin, JW1
Grogan, SP; Hiraoka, K; Lotz, MK; Olee, T1
Beraud, E; Lombardo, D; Mathieu, S; Ristorcelli, E; Verine, A1
Amanda, WZ; Butler-Browne, G; Gluckman, PD; Hui, GZ; Kambadur, R; Lau, HY; Lokireddy, S; McFarlane, C; Mouly, V; Sharma, M; Xiaojia, G1
Dou, F; Lv, D; Miao, F; Shen, Y; Wang, J; Yin, Y; Zhang, J1
Althani, A; Casalbore, P; Cenciarelli, C; Felsani, A; Giannetti, S; Mangiola, A; Marei, HE; Trevisi, G; Zonfrillo, M1

Other Studies

7 other study(ies) available for carbamates and hes1 protein, human

ArticleYear
Notch signaling and ERK activation are important for the osteomimetic properties of prostate cancer bone metastatic cell lines.
    The Journal of biological chemistry, 2004, Jan-30, Volume: 279, Issue:5

    Topics: Basic Helix-Loop-Helix Transcription Factors; Blotting, Western; Bone Neoplasms; Calcium; Carbamates; Cell Differentiation; Cell Line, Tumor; Cell Nucleus; Core Binding Factor Alpha 1 Subunit; Dipeptides; DNA; Enzyme Activation; Homeodomain Proteins; Humans; Immunohistochemistry; Intracellular Signaling Peptides and Proteins; Male; Membrane Proteins; Mitogen-Activated Protein Kinases; Models, Biological; Neoplasm Metastasis; Neoplasm Proteins; Osteoblasts; Prostatic Neoplasms; Protein Binding; Receptors, Notch; Reverse Transcriptase Polymerase Chain Reaction; RNA; RNA, Ribosomal, 18S; Signal Transduction; Time Factors; Transcription Factor HES-1; Transcription Factors

2004
Eosinophilic differentiation is promoted by blockage of Notch signaling with a gamma-secretase inhibitor.
    European journal of immunology, 2005, Volume: 35, Issue:10

    Topics: Amyloid Precursor Protein Secretases; Aspartic Acid Endopeptidases; Basic Helix-Loop-Helix Transcription Factors; Carbamates; Cell Death; Cell Differentiation; Cell Proliferation; Chemotaxis, Leukocyte; Dipeptides; Endopeptidases; Enzyme Inhibitors; Eosinophil Major Basic Protein; Eosinophils; Flow Cytometry; Homeodomain Proteins; Humans; Immunohistochemistry; Receptors, CCR3; Receptors, Chemokine; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Transcription Factor HES-1

2005
Repression of chondrogenesis through binding of notch signaling proteins HES-1 and HEY-1 to N-box domains in the COL2A1 enhancer site.
    Arthritis and rheumatism, 2008, Volume: 58, Issue:9

    Topics: Basic Helix-Loop-Helix Transcription Factors; Blotting, Western; Bone Morphogenetic Protein 6; Carbamates; Cell Cycle Proteins; Cell Differentiation; Cells, Cultured; Chondrogenesis; Chromatin Immunoprecipitation; Collagen Type II; Dipeptides; Fibroblast Growth Factor 2; Homeodomain Proteins; Humans; Mesenchymal Stem Cells; Receptors, Notch; Regulatory Sequences, Nucleic Acid; Repressor Proteins; Reverse Transcriptase Polymerase Chain Reaction; Signal Transduction; Time Factors; Transcription Factor HES-1; Transforming Growth Factor beta3

2008
Essential role of Notch signaling in apoptosis of human pancreatic tumoral cells mediated by exosomal nanoparticles.
    International journal of cancer, 2009, Sep-01, Volume: 125, Issue:5

    Topics: Adenocarcinoma; Amyloid Precursor Protein Secretases; Apoptosis; Basic Helix-Loop-Helix Transcription Factors; Blotting, Western; Carbamates; Caspase 3; Cell Differentiation; Cell Proliferation; Dipeptides; Exosomes; Flow Cytometry; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Homeodomain Proteins; Humans; Mitochondria; Nanoparticles; Pancreatic Neoplasms; PTEN Phosphohydrolase; Receptors, Notch; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Transcription Factor HES-1; Tumor Cells, Cultured

2009
Human myostatin negatively regulates human myoblast growth and differentiation.
    American journal of physiology. Cell physiology, 2011, Volume: 301, Issue:1

    Topics: Adolescent; Animals; Basic Helix-Loop-Helix Transcription Factors; Carbamates; Cell Cycle; Cell Cycle Proteins; Cell Differentiation; Cell Proliferation; Cells, Cultured; Child; Dipeptides; DNA-Binding Proteins; Homeodomain Proteins; Humans; Immunoprecipitation; Mice; Muscle Development; Muscle Fibers, Skeletal; Muscle, Skeletal; Myoblasts; MyoD Protein; Myogenic Regulatory Factors; Myostatin; p21-Activated Kinases; Polymerase Chain Reaction; Receptors, Notch; Recombinant Proteins; Repressor Proteins; Signal Transduction; Smad Proteins; Transcription Factor HES-1; Transforming Growth Factor beta

2011
GSI-I has a better effect in inhibiting hepatocellular carcinoma cell growth than GSI-IX, GSI-X, or GSI-XXI.
    Anti-cancer drugs, 2012, Volume: 23, Issue:7

    Topics: Amyloid Precursor Protein Secretases; Antineoplastic Agents; Basic Helix-Loop-Helix Transcription Factors; Benzodiazepinones; Calcium-Binding Proteins; Carbamates; Carcinoma, Hepatocellular; Cell Line, Tumor; Cell Proliferation; Cell Survival; Dipeptides; Gene Expression Regulation, Neoplastic; Genes, myc; Hep G2 Cells; Homeodomain Proteins; Humans; Intercellular Signaling Peptides and Proteins; Jagged-1 Protein; Liver Neoplasms; Membrane Proteins; Oligopeptides; Receptors, Notch; Serrate-Jagged Proteins; Transcription Factor HES-1

2012
The interference of Notch1 target Hes1 affects cell growth, differentiation and invasiveness of glioblastoma stem cells through modulation of multiple oncogenic targets.
    Oncotarget, 2017, Mar-14, Volume: 8, Issue:11

    Topics: Apoptosis; Benzimidazoles; Brain Neoplasms; Carbamates; Cell Differentiation; Cell Proliferation; Dipeptides; ErbB Receptors; Glioblastoma; Humans; Microtubule-Associated Proteins; Neoplasm Invasiveness; Neoplastic Stem Cells; Phosphorylation; Piperidines; Receptor, Notch1; RNA Interference; RNA, Small Interfering; Signal Transduction; STAT3 Transcription Factor; STAT5 Transcription Factor; Transcription Factor HES-1; Tumor Suppressor Proteins

2017