carbachol has been researched along with diamide in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 3 (75.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bellows, DS; Clarke, ID; Diamandis, P; Dirks, PB; Graham, J; Jamieson, LG; Ling, EK; Sacher, AG; Tyers, M; Ward, RJ; Wildenhain, J | 1 |
| Balduini, W; Cantoni, O; Cattabeni, F | 1 |
| Jope, RS; Li, X; Song, L | 1 |
| Fowler, CJ; Tiger, G | 1 |
4 other study(ies) available for carbachol and diamide
| Article | Year |
|---|---|
Chemical genetics reveals a complex functional ground state of neural stem cells.
Topics: Animals; Cell Survival; Cells, Cultured; Mice; Molecular Structure; Neoplasms; Neurons; Pharmaceutical Preparations; Sensitivity and Specificity; Stem Cells | 2007 |
Modulation of muscarinic receptor-stimulated phosphoinositide breakdown by sulfhydryl group modification is a general response in different rat brain regions and depends on the stage of brain development.
Topics: Aging; Animals; Animals, Newborn; Brain; Carbachol; Cerebral Cortex; Corpus Striatum; Diamide; Dithionitrobenzoic Acid; Ethylmaleimide; Hippocampus; In Vitro Techniques; Male; Muscarinic Agonists; Organ Specificity; Phosphatidylinositols; Rats; Rats, Sprague-Dawley; Receptors, Muscarinic; Sulfhydryl Reagents | 1996 |
Glutathione depletion exacerbates impairment by oxidative stress of phosphoinositide hydrolysis, AP-1, and NF-kappaB activation by cholinergic stimulation.
Topics: Analysis of Variance; Base Sequence; Binding Sites; Buthionine Sulfoximine; Carbachol; Consensus Sequence; Diamide; Glutathione; GTP-Binding Proteins; Humans; Hydrogen Peroxide; Ionomycin; Neuroblastoma; NF-kappa B; Oligodeoxyribonucleotides; Oxidative Stress; Phosphatidylinositols; Second Messenger Systems; Sodium Fluoride; Transcription Factor AP-1; Tumor Cells, Cultured; Vanadates | 1998 |
The sulphydryl oxidizing reagent diamide affects phosphoinositide-mediated signal transduction: implications for the pathogenesis of Alzheimer's disease.
Topics: Alzheimer Disease; Animals; Blood Platelets; Brain; Calcium Signaling; Carbachol; Diamide; Dithiothreitol; Humans; Hydrogen Peroxide; Hydrolysis; Inositol 1,4,5-Trisphosphate; Norepinephrine; Oxidants; Oxidative Stress; Phosphatidylinositols; Rats; Serotonin; Sulfhydryl Reagents; Thrombin | 1998 |