bromocriptine has been researched along with u 0126 in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bellows, DS; Clarke, ID; Diamandis, P; Dirks, PB; Graham, J; Jamieson, LG; Ling, EK; Sacher, AG; Tyers, M; Ward, RJ; Wildenhain, J | 1 |
| Baker, RE; Elsholtz, HP; Liu, JC; Sun, C; Sundmark, VC | 1 |
| Arita, J; Ishida, M; Izawa, M; Mitsui, T | 1 |
3 other study(ies) available for bromocriptine and u 0126
| Article | Year |
|---|---|
Chemical genetics reveals a complex functional ground state of neural stem cells.
Topics: Animals; Cell Survival; Cells, Cultured; Mice; Molecular Structure; Neoplasms; Neurons; Pharmaceutical Preparations; Sensitivity and Specificity; Stem Cells | 2007 |
Activation of Go-coupled dopamine D2 receptors inhibits ERK1/ERK2 in pituitary cells. A key step in the transcriptional suppression of the prolactin gene.
Topics: Animals; Binding Sites; Bromocriptine; Butadienes; Cells, Cultured; Cloning, Molecular; Dopamine Agonists; Enzyme Activation; Enzyme Inhibitors; Gene Expression Regulation; Green Fluorescent Proteins; Immunoblotting; Inhibitory Concentration 50; Luminescent Proteins; MAP Kinase Signaling System; Microscopy, Confocal; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Models, Chemical; Mutagenesis, Site-Directed; Nitriles; Pertussis Toxin; Phosphorylation; Pituitary Gland; Plasmids; Prolactin; Promoter Regions, Genetic; Rats; Rats, Sprague-Dawley; Receptors, Dopamine D2; RNA; Time Factors; Transcription, Genetic | 2002 |
Absence of ligand-independent transcriptional activation of the estrogen receptor via the estrogen response element in pituitary lactotrophs in primary culture.
Topics: Adenoviridae; Animals; Bromocriptine; Butadienes; Cell Line, Tumor; Cells, Cultured; Colforsin; Epidermal Growth Factor; Estradiol; Estrogen Antagonists; Female; Fulvestrant; Genes, Reporter; Insulin-Like Growth Factor I; Integrases; Isoquinolines; Lactotrophs; Ligands; Luciferases; Nitriles; Pituitary Gland, Anterior; Prolactin; Promoter Regions, Genetic; Protein Kinase Inhibitors; Rats; Rats, Wistar; Receptors, Estrogen; Response Elements; Sulfonamides; Thymidine Kinase; Transcriptional Activation; Transduction, Genetic | 2010 |