benzofurans and 3-3--dipropyl-2-2--thiadicarbocyanine

benzofurans has been researched along with 3-3--dipropyl-2-2--thiadicarbocyanine* in 1 studies

Other Studies

1 other study(ies) available for benzofurans and 3-3--dipropyl-2-2--thiadicarbocyanine

Article	Year
[Calcium antagonists nicardipine and verapamil suppress Ca(2+) entry and activate Na(+) entry into platelets]. Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova, 2000, Volume: 86, Issue:9 In micromolar concentrations both antagonists suppressed CA2+ entry and simultaneously elevate the agonist-induced plasma membrane depolarization due to Na+ inward current via these channels. Potentiation by nicardipine of the Na+ current induced by the platelet-activating factor, was revealed. Both antagonists caused plasma membrane depolarization suppressed by Na+ and Ca2+ ions. The depolarization disappeared after substitution of NaCl by an isotonic solution of choline chloride. The antagonists nicardipine and verapamil seem to modulate the platelet receptor-operated channels suppressing Ca2+ entry and elevating Na+ current via these channels. Topics: Benzofurans; Benzothiazoles; Blood Platelets; Calcium; Calcium Channel Blockers; Carbocyanines; Ethers, Cyclic; Fluorescent Dyes; Humans; In Vitro Techniques; Nicardipine; Sodium; Sodium Channels; Verapamil	2000

Article

Year

[Calcium antagonists nicardipine and verapamil suppress Ca(2+) entry and activate Na(+) entry into platelets].

Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova, 2000, Volume: 86, Issue:9

In micromolar concentrations both antagonists suppressed CA2+ entry and simultaneously elevate the agonist-induced plasma membrane depolarization due to Na+ inward current via these channels. Potentiation by nicardipine of the Na+ current induced by the platelet-activating factor, was revealed. Both antagonists caused plasma membrane depolarization suppressed by Na+ and Ca2+ ions. The depolarization disappeared after substitution of NaCl by an isotonic solution of choline chloride. The antagonists nicardipine and verapamil seem to modulate the platelet receptor-operated channels suppressing Ca2+ entry and elevating Na+ current via these channels.

Topics: Benzofurans; Benzothiazoles; Blood Platelets; Calcium; Calcium Channel Blockers; Carbocyanines; Ethers, Cyclic; Fluorescent Dyes; Humans; In Vitro Techniques; Nicardipine; Sodium; Sodium Channels; Verapamil

2000