benazepril and plerixafor

benazepril has been researched along with plerixafor* in 1 studies

Other Studies

1 other study(ies) available for benazepril and plerixafor

Article	Year
Effects of benazepril on functional activity of endothelial progenitor cells from hypertension patients. Clinical and experimental hypertension (New York, N.Y. : 1993), 2014, Volume: 36, Issue:8 The effect of angiotensin-converting enzyme inhibitors on hypertension patients regarding endothelial progenitor cell (EPC) functions is poorly understood. The aim of this study was to investigate the effects of benazepril on the proliferation, adhesion and migration capacity of EPCs and its possible mechanism. The functions of EPCs from hypertension patients were obviously reduced compared with control group, and this could be improved by benazepril in a dose-dependent manner, whereas this improvement were obviously blocked when AMD3100 were used together. Therefore, benazepril could obviously improve functions of EPCs from hypertension patients, and the potential mechanism may be related to SDF-1/CXCR4 axis. Topics: Angiotensin-Converting Enzyme Inhibitors; Antihypertensive Agents; Benzazepines; Benzylamines; Case-Control Studies; Cell Adhesion; Cell Movement; Cell Proliferation; Cells, Cultured; Chemokine CXCL12; Cyclams; Endothelial Progenitor Cells; Gene Expression; Heterocyclic Compounds; Humans; Hypertension; Receptors, CXCR4; RNA, Messenger	2014

Article

Year

Effects of benazepril on functional activity of endothelial progenitor cells from hypertension patients.

Clinical and experimental hypertension (New York, N.Y. : 1993), 2014, Volume: 36, Issue:8

The effect of angiotensin-converting enzyme inhibitors on hypertension patients regarding endothelial progenitor cell (EPC) functions is poorly understood. The aim of this study was to investigate the effects of benazepril on the proliferation, adhesion and migration capacity of EPCs and its possible mechanism. The functions of EPCs from hypertension patients were obviously reduced compared with control group, and this could be improved by benazepril in a dose-dependent manner, whereas this improvement were obviously blocked when AMD3100 were used together. Therefore, benazepril could obviously improve functions of EPCs from hypertension patients, and the potential mechanism may be related to SDF-1/CXCR4 axis.

Topics: Angiotensin-Converting Enzyme Inhibitors; Antihypertensive Agents; Benzazepines; Benzylamines; Case-Control Studies; Cell Adhesion; Cell Movement; Cell Proliferation; Cells, Cultured; Chemokine CXCL12; Cyclams; Endothelial Progenitor Cells; Gene Expression; Heterocyclic Compounds; Humans; Hypertension; Receptors, CXCR4; RNA, Messenger

2014