bay-k-8644 has been researched along with remifentanil in 1 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 1 (100.00) | 2.80 |
Authors | Studies |
---|---|
Chen, L; Chen, Z; Gao, X; Li, Z; Lin, C; Liu, T; Qi, F; Tang, H; Wang, L; Wang, ZJ; Zhang, X | 1 |
1 other study(ies) available for bay-k-8644 and remifentanil
Article | Year |
---|---|
Ketamine reduces remifentanil-induced postoperative hyperalgesia mediated by CaMKII-NMDAR in the primary somatosensory cerebral cortex region in mice.
Topics: 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; Analgesics; Analgesics, Opioid; Animals; Benzylamines; Blotting, Western; Calcium Channel Agonists; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Fluorescent Antibody Technique; Hyperalgesia; Ketamine; Mice; Pain Threshold; Pain, Postoperative; Postoperative Complications; Protein Kinase Inhibitors; Receptors, N-Methyl-D-Aspartate; Remifentanil; Somatosensory Cortex; Sulfonamides | 2020 |