baclofen has been researched along with n(6)-cyclohexyladenosine in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (25.00) | 18.7374 |
1990's | 2 (50.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Fowler, JC | 1 |
Cavalla, D; Neff, NH; Wojcik, WJ | 1 |
Dar, MS | 1 |
Brust, TB; Cayabyab, FS; MacVicar, BA | 1 |
4 other study(ies) available for baclofen and n(6)-cyclohexyladenosine
Article | Year |
---|---|
Escape from inhibition of synaptic transmission during in vitro hypoxia and hypoglycemia in the hippocampus.
Topics: Adenosine; Animals; Baclofen; Brain Ischemia; Cell Hypoxia; Electric Stimulation; Electrophysiology; Hippocampus; Hypoglycemia; In Vitro Techniques; Male; Rats; Rats, Inbred Strains; Synapses; Synaptic Transmission | 1992 |
Co-localized adenosine A1 and gamma-aminobutyric acid B (GABAB) receptors of cerebellum may share a common adenylate cyclase catalytic unit.
Topics: Adenosine; Adenylyl Cyclases; Animals; Baclofen; Cerebellum; GTP Phosphohydrolases; Isoproterenol; Kinetics; Macromolecular Substances; Male; Norepinephrine; Phenylisopropyladenosine; Rats; Rats, Inbred Strains; Receptors, Cell Surface; Receptors, GABA-A; Receptors, Purinergic; Synaptic Membranes | 1985 |
Mouse cerebellar GABAB participation in the expression of acute ethanol-induced ataxia and in its modulation by the cerebellar adenosinergic A1 system.
Topics: Adenosine; Adenylate Cyclase Toxin; Animals; Ataxia; Baclofen; Behavior, Animal; Cerebellum; Dose-Response Relationship, Drug; Ethanol; GABA Agonists; GTP-Binding Proteins; Male; Mice; Mice, Inbred Strains; Microinjections; Motor Activity; Muscimol; Pertussis Toxin; Receptors, GABA-B; Receptors, Purinergic P1; Virulence Factors, Bordetella | 1996 |
C-Jun N-terminal kinase regulates adenosine A1 receptor-mediated synaptic depression in the rat hippocampus.
Topics: Adenosine; Adenosine A1 Receptor Agonists; Adenosine A1 Receptor Antagonists; Animals; Animals, Newborn; Baclofen; Drug Interactions; Electric Stimulation; Enzyme Inhibitors; Excitatory Postsynaptic Potentials; GABA Agonists; Gene Expression; Hippocampus; Hypoxia; In Vitro Techniques; JNK Mitogen-Activated Protein Kinases; Long-Term Synaptic Depression; Rats; Rats, Sprague-Dawley; Receptor, Adenosine A1; Time Factors; Xanthines | 2007 |