atorvastatin has been researched along with aica ribonucleotide in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, Z; DeFea, K; Fu, Y; Hsu, PH; Pan, S; Peng, IC; Shyy, JY; Su, MI; Sun, W; Tsai, MD; Zhu, Y | 1 |
| Chen, Z; Jia, F; Lu, G; Wu, C | 2 |
3 other study(ies) available for atorvastatin and aica ribonucleotide
| Article | Year |
|---|---|
AMP-activated protein kinase functionally phosphorylates endothelial nitric oxide synthase Ser633.
Topics: Acetyl-CoA Carboxylase; Adiponectin; Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Atorvastatin; Cattle; Cells, Cultured; Chromatography, Liquid; Endothelial Cells; Enzyme Activation; Heptanoic Acids; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Mice; Mice, Inbred C57BL; Mice, Knockout; Mutation; Nanotechnology; Nitric Oxide; Nitric Oxide Synthase Type III; Phosphorylation; Protein Kinase Inhibitors; Protein Processing, Post-Translational; Pyrazoles; Pyrimidines; Pyrroles; Ribonucleotides; RNA Interference; RNA, Small Interfering; Serine; Signal Transduction; Stress, Mechanical; Tandem Mass Spectrometry; Time Factors; Transfection | 2009 |
AMP-activated protein kinase inhibits homocysteine-induced dysfunction and apoptosis in endothelial progenitor cells.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Apoptosis; Atorvastatin; Cell Movement; Cell Proliferation; Cells, Cultured; Down-Regulation; Endothelial Cells; Heptanoic Acids; Homocysteine; Humans; NADPH Oxidase 4; NADPH Oxidases; Nitric Oxide; Nitric Oxide Synthase Type III; Phosphorylation; Pyrroles; Reactive Oxygen Species; Ribonucleotides; Stem Cells; Up-Regulation | 2011 |
Atorvastatin inhibits homocysteine-induced endoplasmic reticulum stress through activation of AMP-activated protein kinase.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Anticholesteremic Agents; Atherosclerosis; Atorvastatin; Blotting, Western; Dose-Response Relationship, Drug; Endoplasmic Reticulum Stress; Heptanoic Acids; Homocysteine; Human Umbilical Vein Endothelial Cells; Humans; Lipids; Mice; Polymerase Chain Reaction; Pyrroles; Ribonucleotides; Time Factors | 2012 |