aspirin and butaprost
aspirin has been researched along with butaprost in 3 studies
Research
Studies (3)
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 2 (66.67) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors
Authors | Studies |
---|---|
Akaike, A; Banno, Y; Fushimi, K; Nakashima, S; Shimizu, K; Takigawa, M | 1 |
Alobid, I; Gabasa, M; Machado-Carvalho, L; Martín, M; Mullol, J; Picado, C; Pujols, L; Roca-Ferrer, J; Torres, R | 1 |
Alobid, I; Machado-Carvalho, L; Mullol, J; Perez-Gonzalez, M; Picado, C; Pujols, L; Roca-Ferrer, J; Torres, R | 1 |
Other Studies
3 other study(ies) available for aspirin and butaprost
Article | Year |
---|---|
Implication of prostaglandin E(2) in TNF-alpha-induced release of m-calpain from HCS-2/8 chondrocytes. Inhibition of m-calpain release by NSAIDs.
Topics: Alprostadil; Anti-Inflammatory Agents, Non-Steroidal; Apoptosis; Aspirin; Calpain; Cell Line, Tumor; Chondrocytes; Dibenz(b,f)(1,4)oxazepine-10(11H)-carboxylic acid, 8-chloro-, 2-acetylhydrazide; Diclofenac; Dinoprostone; Humans; Nitrobenzenes; Phenylpropionates; Prostaglandin Antagonists; Sulfonamides; Tumor Necrosis Factor-alpha; Up-Regulation; Xanthones | 2004 |
Low E-prostanoid 2 receptor levels and deficient induction of the IL-1β/IL-1 type I receptor/COX-2 pathway: Vicious circle in patients with aspirin-exacerbated respiratory disease.
Topics: Adult; Aged; Alprostadil; Aspirin; Asthma, Aspirin-Induced; Cells, Cultured; Cyclooxygenase 2; Dinoprostone; Female; Fibroblasts; Humans; Interleukin-1beta; Intramolecular Oxidoreductases; Male; Middle Aged; Nasal Mucosa; Nasal Polyps; Prostaglandin-E Synthases; Receptors, Interleukin-1 Type I; Receptors, Prostaglandin E, EP2 Subtype; RNA, Messenger | 2016 |
Altered expression and signalling of EP2 receptor in nasal polyps of AERD patients: role in inflammation and remodelling.
Topics: Alprostadil; Anti-Inflammatory Agents, Non-Steroidal; Aspirin; Cell Proliferation; Cyclic AMP; Dinoprostone; Disease Progression; Down-Regulation; Female; Fibroblasts; Granulocyte-Macrophage Colony-Stimulating Factor; Humans; Inflammation; Male; Middle Aged; Nasal Mucosa; Nasal Polyps; Receptors, Prostaglandin E; Respiratory Tract Diseases; Signal Transduction | 2016 |