asialo-gm1-ganglioside and romurtide

asialo-gm1-ganglioside has been researched along with romurtide* in 1 studies

Other Studies

1 other study(ies) available for asialo-gm1-ganglioside and romurtide

Article	Year
Suppression of Sendai virus growth by treatment with N alpha-acetylmuramyl-L-alanyl-D-isoglutaminyl-N epsilon-stearoyl-L-lysine in mice. Vaccine, 1987, Volume: 5, Issue:4 Mice that received N alpha-acetylmuramyl-L-alanyl-D-isoglutaminyl-N epsilon-stearoyl-L-lysine [MDP-Lys (L18)] were resistant to Sendai virus infection. In these protected mice, a significant growth inhibition of the virus was confirmed repeatedly at 10(0.2) to 10(0.4) of haemadsorbing units at an early non-specific phase but not at a late virus-eliminating phase of the infection. Virus growth was enhanced by treatment with silica but not by treatment with anti-asialo GM1 serum in MDP-Lys (L18)-treated mice. Peritoneal adherent cells activated by MDP-Lys(L18) showed an enhanced uptake and ability to inactivate Sendai virus in vitro. Excess interferon production in MDP-Lys (L18)-treated mice was seen on day 1 but not on days 2 to 7 of the infection. The possible role of macrophages and interferon in providing non-specific protection against Sendai virus in the MDP-Lys (L18)-treated mice is discussed. Topics: Acetylmuramyl-Alanyl-Isoglutamine; Animals; G(M1) Ganglioside; Glycosphingolipids; Immune Sera; Interferons; Lung; Male; Mice; Mice, Inbred BALB C; Parainfluenza Virus 1, Human; Paramyxoviridae Infections; Silicon Dioxide	1987

Article

Year

Suppression of Sendai virus growth by treatment with N alpha-acetylmuramyl-L-alanyl-D-isoglutaminyl-N epsilon-stearoyl-L-lysine in mice.

Vaccine, 1987, Volume: 5, Issue:4

Mice that received N alpha-acetylmuramyl-L-alanyl-D-isoglutaminyl-N epsilon-stearoyl-L-lysine [MDP-Lys (L18)] were resistant to Sendai virus infection. In these protected mice, a significant growth inhibition of the virus was confirmed repeatedly at 10(0.2) to 10(0.4) of haemadsorbing units at an early non-specific phase but not at a late virus-eliminating phase of the infection. Virus growth was enhanced by treatment with silica but not by treatment with anti-asialo GM1 serum in MDP-Lys (L18)-treated mice. Peritoneal adherent cells activated by MDP-Lys(L18) showed an enhanced uptake and ability to inactivate Sendai virus in vitro. Excess interferon production in MDP-Lys (L18)-treated mice was seen on day 1 but not on days 2 to 7 of the infection. The possible role of macrophages and interferon in providing non-specific protection against Sendai virus in the MDP-Lys (L18)-treated mice is discussed.

Topics: Acetylmuramyl-Alanyl-Isoglutamine; Animals; G(M1) Ganglioside; Glycosphingolipids; Immune Sera; Interferons; Lung; Male; Mice; Mice, Inbred BALB C; Parainfluenza Virus 1, Human; Paramyxoviridae Infections; Silicon Dioxide

1987