arsenic trioxide has been researched along with cytochrome c-t in 22 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 15 (68.18) | 29.6817 |
| 2010's | 7 (31.82) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| An, S; Bae, IJ; Gwak, HS; Hong, SI; Lee, HC; Lee, SH; Park, IC; Park, MJ; Rhee, CH; Seo, KM; Woo, SH | 1 |
| Almasan, A; Chen, J; Chen, Q; Jiang, C; Jin, H; Shi, Y; Tang, H; Tian, C; Zheng, Y | 1 |
| Akay, C; Gazitt, Y; Thomas, C | 1 |
| Karlsson, J; Påhlman, S; Pörn-Ares, I; Øra, I | 1 |
| Wang, W; Zhang, R; Zhang, XH; Zhu, ZL | 1 |
| Chen, YY; Guo, QS; Hu, QS; Kang, XL; Li, H; Lu, Y; Shi, GY; Tang, XM; Wang, XJ; Yang, J; Yi, J | 1 |
| Huang, X; Ma, A; Ma, X; Qiao, D; Tian, X; Yan, F | 1 |
| Ma, AD; Ma, XD; Qiao, DF; Tian, XM; Yan, F | 1 |
| Brdiczka, D; Israelson, A; Sheu, SS; Shoshan-Barmatz, V | 1 |
| Baysan, A; Gollapudi, S; Gupta, S; Su, H; Yel, L | 1 |
| Chen, JP; Cong, YW; Dong, B; Fu, YL; Huang, R; Liang, YG; Liu, ZY; Mao, BZ; Shan, YJ; Wang, SQ; Xiao, H; Yu, ZY; Zhao, QS; Zhao, ZH | 1 |
| Bismar, TA; Diaz, Z; Laurenzana, A; Mann, KK; Miller, WH; Schipper, HM | 1 |
| Chen, Q; Gao, P; Jin, H; Tian, C; Wang, X; Yue, W; Zheng, Y | 1 |
| Chen, WC; Chiu, HW; Guo, HR; Ho, SY; Lai, CS; Wang, YJ | 1 |
| Hong, Y; Li, S; Liu, P; Piao, F; Wang, Y; Zhao, Y | 1 |
| Chen, MF; Chen, MJ; Hu, DN; Yang, PY; Ye, YZ | 1 |
| Chung, JY; Han, SH; Joo, BS; Kim, JM; Kim, JY; Kim, WR; Kim, YJ; Lee, SG; Park, JE; Yoo, KS; Yoo, YH | 1 |
| Tang, X; Wang, C; Xiao, G; Yao, C | 1 |
| Abreu e Lima, RS; Curti, C; de Thé, H; dos Santos, GA; Falcão, RP; Gimenes-Teixeira, HL; Lima, AS; Lucena-Araujo, AR; Nasr, R; Pandolfi, PP; Pestana, CR; Rego, EM; Rodrigues, FP; Santana-Lemos, BA; Scheucher, PS; Thomé, CH; Uyemura, SA | 1 |
| Kumar, S; Tchounwou, PB; Yedjou, CG | 1 |
| Chen, XY; Fan, XY; Jiang, FL; Liu, Y; Liu, YJ; Zhong, HM | 1 |
| Afzal, M; Anis, E; Firdaus, F; Hossain, MM; Waseem, M; Zafeer, MF | 1 |
1 review(s) available for arsenic trioxide and cytochrome c-t
| Article | Year |
|---|---|
The voltage-dependent anion channel (VDAC): function in intracellular signalling, cell life and cell death.
Topics: Animals; Antineoplastic Agents; Apoptosis; Arsenic Trioxide; Arsenicals; bcl-2-Associated X Protein; Calcium; Cytochromes c; Humans; Ion Channel Gating; Mitochondria; Mitochondrial Membranes; Models, Molecular; Neoplasms; Oxides; Permeability; Protein Conformation; Protein Folding; Protein Isoforms; Proto-Oncogene Proteins c-bcl-2; Ruthenium Red; Signal Transduction; Voltage-Dependent Anion Channels | 2006 |
21 other study(ies) available for arsenic trioxide and cytochrome c-t
| Article | Year |
|---|---|
Tetraarsenic oxide induces apoptosis in U937 leukemic cells through a reactive oxygen species-dependent pathway.
Topics: Apoptosis; Arsenic; Arsenic Trioxide; Arsenicals; Caspase 3; Caspases; Catalase; Cytochromes c; Dose-Response Relationship, Drug; Enzyme Activation; Humans; Hydrogen Peroxide; Leukemia; Membrane Potentials; Mitochondria; Oxides; Reactive Oxygen Species; Time Factors; U937 Cells | 2003 |
Essential role of the voltage-dependent anion channel (VDAC) in mitochondrial permeability transition pore opening and cytochrome c release induced by arsenic trioxide.
Topics: Animals; Annexin A5; Arsenic Trioxide; Arsenicals; Cytochromes c; Flow Cytometry; Humans; Intracellular Membranes; Ion Channel Gating; Membrane Potentials; Mice; Mice, Inbred BALB C; Mitochondria; Mitochondria, Liver; Multiple Myeloma; Oxides; Permeability; Porins; Proteolipids; Tumor Cells, Cultured; Voltage-Dependent Anion Channels | 2004 |
Arsenic trioxide and paclitaxel induce apoptosis by different mechanisms.
Topics: Apoptosis; Apoptosis Inducing Factor; Apoptosis Regulatory Proteins; Arsenic Trioxide; Arsenicals; BH3 Interacting Domain Death Agonist Protein; Carrier Proteins; Caspases; Cell Line; Cell Nucleus; Chromatin; Cytochromes c; Enzyme Activation; Flavoproteins; Flow Cytometry; Gene Expression Regulation; Humans; Intracellular Membranes; Membrane Glycoproteins; Membrane Proteins; Mitochondria; Oxides; Paclitaxel; Phosphoserine; Protein Transport; Proto-Oncogene Proteins c-bcl-2; Time Factors; TNF-Related Apoptosis-Inducing Ligand; Tumor Necrosis Factor-alpha | 2004 |
Arsenic trioxide-induced death of neuroblastoma cells involves activation of Bax and does not require p53.
Topics: Antineoplastic Agents; Apoptosis; Arsenic Trioxide; Arsenicals; bcl-2-Associated X Protein; Blotting, Western; Calpain; Carboplatin; Caspase 3; Caspases; Cell Line, Tumor; Cytochromes c; Dose-Response Relationship, Drug; Doxorubicin; Drug Resistance, Neoplasm; Etoposide; Humans; Mutation; Neuroblastoma; Oxides; Proto-Oncogene Proteins c-bcl-2; Time Factors; Tumor Suppressor Protein p53; Vincristine | 2004 |
[The mechanism of arsenic trioxide-inducing apoptosis of K562 cells].
Topics: Antineoplastic Agents; Apoptosis; Arsenic Trioxide; Arsenicals; bcl-X Protein; Caspase 3; Caspases; Cell Cycle; Cell Proliferation; Cytochromes c; Fusion Proteins, bcr-abl; Humans; K562 Cells; Oxides; Proto-Oncogene Proteins c-bcl-2 | 2004 |
Anthraquinones sensitize tumor cells to arsenic cytotoxicity in vitro and in vivo via reactive oxygen species-mediated dual regulation of apoptosis.
Topics: Animals; Anthraquinones; Antioxidants; Apoptosis; Arsenic Trioxide; Arsenicals; Caspase 3; Caspase 9; Caspases; Cell Line, Tumor; Cytochromes c; Emodin; Enzyme Activation; Humans; Intracellular Membranes; Mice; Mitochondria; Neoplasm Transplantation; Neoplasms; NF-kappa B; Oxides; Phorbol Esters; Reactive Oxygen Species; Signal Transduction | 2004 |
mCICR is required for As2O3-induced permeability transition pore opening and cytochrome c release from mitochondria.
Topics: Animals; Apoptosis; Arsenic Trioxide; Arsenicals; Calcium Signaling; Cytochromes c; Female; In Vitro Techniques; Ion Channels; Kinetics; Mitochondria, Liver; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Oxides; Rats; Rats, Wistar | 2005 |
[Mechanism of opening of mitochondrial permeability transition pore induced by arsenic trioxide].
Topics: Animals; Apoptosis; Arsenic Trioxide; Arsenicals; Calcium; Cytochromes c; Female; In Vitro Techniques; Mitochondria, Liver; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Oxides; Rats; Rats, Wistar | 2006 |
Arsenic trioxide induces apoptosis via the mitochondrial pathway by upregulating the expression of Bax and Bim in human B cells.
Topics: Antineoplastic Agents; Apoptosis; Apoptosis Inducing Factor; Apoptosis Regulatory Proteins; Arsenic Trioxide; Arsenicals; B-Lymphocytes; bcl-2-Associated X Protein; Bcl-2-Like Protein 11; Cytochromes c; Cytoplasm; Humans; Leukemia, Promyelocytic, Acute; Male; Membrane Potentials; Membrane Proteins; Mitochondria; Oxidative Stress; Oxides; Proto-Oncogene Proteins; Up-Regulation | 2007 |
Melissoidesin G, a diterpenoid purified from Isodon melissoides, induces leukemic-cell apoptosis through induction of redox imbalance and exhibits synergy with other anticancer agents.
Topics: Antineoplastic Agents; Apoptosis; Arsenic Trioxide; Arsenicals; Caspases; Cytochromes c; Diterpenes; Glutathione; Humans; Isodon; Leukemia; Mitochondria; Molecular Structure; Oxidation-Reduction; Oxides; Phytotherapy; Tumor Cells, Cultured | 2007 |
Trolox enhances the anti-lymphoma effects of arsenic trioxide, while protecting against liver toxicity.
Topics: Animals; Antineoplastic Combined Chemotherapy Protocols; Antioxidants; Apoptosis; Arsenic Trioxide; Arsenicals; Cell Line, Tumor; Chromans; Cytochromes c; Drug Synergism; Humans; Liver; Lymphoma; Membrane Potential, Mitochondrial; Mice; Neoplasm Metastasis; Neoplasm Transplantation; Oxides | 2007 |
Redox status of thioredoxin-1 (TRX1) determines the sensitivity of human liver carcinoma cells (HepG2) to arsenic trioxide-induced cell death.
Topics: Animals; Arsenic Trioxide; Arsenicals; Binding Sites; Carcinoma, Hepatocellular; Caspases; Cell Death; Cell Line, Tumor; Cytochromes c; Dinitrochlorobenzene; Drug Screening Assays, Antitumor; Enzyme Activation; Humans; Liver Neoplasms; Mice; Mitochondria; Mitochondrial Swelling; Mutant Proteins; NADP; Oxidation-Reduction; Oxides; Reactive Oxygen Species; Recombinant Proteins; RNA Interference; Thioredoxins | 2008 |
Combination treatment with arsenic trioxide and irradiation enhances apoptotic effects in U937 cells through increased mitotic arrest and ROS generation.
Topics: Antineoplastic Agents; Apoptosis; Arsenic Trioxide; Arsenicals; Caspases; Cell Cycle; Cell Division; Cyclin A; Cyclin B; Cytochromes c; Drug Screening Assays, Antitumor; G2 Phase; Humans; Mitochondrial Membranes; Mitosis; Oxides; Radiation, Ionizing; Reactive Oxygen Species; Tumor Cells, Cultured; Tumor Suppressor Protein p53; U937 Cells | 2009 |
Subchronic exposure to arsenic decreased Sdha expression in the brain of mice.
Topics: 8-Hydroxy-2'-Deoxyguanosine; Animals; Antioxidants; Arsenic Trioxide; Arsenicals; Ascorbic Acid; ATP Synthetase Complexes; Brain; Computational Biology; Cytochromes c; Deoxyguanosine; Dose-Response Relationship, Drug; Electron Transport; Electron Transport Chain Complex Proteins; Electron Transport Complex III; Electron Transport Complex IV; Gene Expression Profiling; Gene Expression Regulation; Growth Inhibitors; Mice; Oligonucleotide Array Sequence Analysis; Oxides; Succinate Dehydrogenase; Taurine | 2009 |
Arsenic trioxide induces apoptosis in uveal melanoma cells through the mitochondrial pathway.
Topics: Antineoplastic Agents; Apoptosis; Arsenic Trioxide; Arsenicals; bcl-2-Associated X Protein; Caspase 3; Caspase 9; Cell Line, Tumor; Cytochromes c; DNA Fragmentation; Dose-Response Relationship, Drug; Eye Neoplasms; Humans; Melanoma; Mitochondria; Oxides; Phytotherapy; Proto-Oncogene Proteins c-bcl-2; Signal Transduction | 2010 |
Arsenic trioxide-induced apoptosis in TM4 Sertoli cells: the potential involvement of p21 expression and p53 phosphorylation.
Topics: Animals; Apoptosis; Arsenic Trioxide; Arsenicals; Blotting, Western; Caspases; Cell Cycle; Cell Line; Cytochromes c; Male; Membrane Potential, Mitochondrial; Mice; Mitochondria; Oncogene Protein p21(ras); Oxides; Phosphorylation; Reactive Oxygen Species; Sertoli Cells; Tumor Suppressor Protein p53 | 2011 |
Potentiation of arsenic trioxide-induced apoptosis by 8-bromo-7-methoxychrysin in human leukemia cells involves depletion of intracellular reduced glutathione.
Topics: Acetylcysteine; Antineoplastic Agents; Apoptosis; Arsenic Trioxide; Arsenicals; bcl-2-Associated X Protein; bcl-X Protein; Blotting, Western; Cell Survival; Cytochromes c; Dose-Response Relationship, Drug; Drug Synergism; Flavonoids; Glutathione; HL-60 Cells; Humans; Intracellular Space; Jurkat Cells; Leukemia; Mitochondria; Oxides; Phosphorylation; Proto-Oncogene Proteins c-akt; Time Factors; U937 Cells; X-Linked Inhibitor of Apoptosis Protein | 2011 |
(+)α-Tocopheryl succinate inhibits the mitochondrial respiratory chain complex I and is as effective as arsenic trioxide or ATRA against acute promyelocytic leukemia in vivo.
Topics: alpha-Tocopherol; Animals; Antineoplastic Agents; Antioxidants; Apoptosis; Arsenic Trioxide; Arsenicals; Caspases; Cell Line, Tumor; Cytochromes c; Disease Models, Animal; Electron Transport Complex I; Electron Transport Complex II; Humans; Leukemia, Promyelocytic, Acute; Membrane Potential, Mitochondrial; Mice; Mice, Transgenic; Mitochondria; Neoplastic Stem Cells; Oncogene Proteins, Fusion; Oxides; Protein Stability; Rats; Reactive Oxygen Species; Transplantation, Isogeneic; Tretinoin | 2012 |
Arsenic trioxide induces oxidative stress, DNA damage, and mitochondrial pathway of apoptosis in human leukemia (HL-60) cells.
Topics: Antineoplastic Agents; Apoptosis; Apoptosis Regulatory Proteins; Arsenic Trioxide; Arsenicals; bcl-2-Associated X Protein; Caspase 3; Cytochromes c; DNA Damage; Drug Screening Assays, Antitumor; Glutathione; HL-60 Cells; Humans; Leukemia, Promyelocytic, Acute; Lipid Peroxidation; Membrane Potential, Mitochondrial; Mitochondria; Oxidative Stress; Oxides; Protein Transport; Single-Cell Analysis | 2014 |
Oxidative stress-mediated intrinsic apoptosis in human promyelocytic leukemia HL-60 cells induced by organic arsenicals.
Topics: Apoptosis; Arsenic Trioxide; Arsenicals; Caspase 3; Cell Proliferation; Cytochromes c; Gene Expression Regulation, Neoplastic; HL-60 Cells; Humans; Leukemia, Promyelocytic, Acute; MCF-7 Cells; Membrane Potential, Mitochondrial; Oxidative Stress; Oxides; Reactive Oxygen Species; Signal Transduction | 2016 |
Ellagic acid mitigates arsenic-trioxide-induced mitochondrial dysfunction and cytotoxicity in SH-SY5Y cells.
Topics: Antineoplastic Agents; Antioxidants; Apoptosis; Arsenic Trioxide; Arsenicals; Cell Line, Tumor; Cell Survival; Comet Assay; Cytochromes c; DNA Fragmentation; Ellagic Acid; Humans; Membrane Potential, Mitochondrial; Mitochondria; Mitochondrial Dynamics; Nerve Tissue Proteins; Neurons; Neuroprotective Agents; Osmolar Concentration; Oxidative Stress; Oxides; Reactive Oxygen Species | 2018 |