apnea has been researched along with methacholine chloride in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Houtz, PK; Lai, YL; Lai-Fook, SJ | 1 |
Xu, F; Zang, N; Zhao, L; Zhuang, J | 1 |
Abel, PW; Aggarwal, A; Bockman, CS; Deodhar, M; Hallgren, J; Iyer, SH; Kansal, V; Matthews, SA; Netzel, L; Oldenburg, PJ; Simeone, KA; Simeone, TA | 1 |
Abel, PW; Aggarwal, A; Hallgren, J; Iyer, SH; Simeone, KA; Simeone, TA; Warren, TJ | 1 |
4 other study(ies) available for apnea and methacholine chloride
Article | Year |
---|---|
End-expiratory and tidal volumes measured in conscious mice using single projection x-ray images.
Topics: Administration, Inhalation; Aerosols; Airway Resistance; Anesthetics, Dissociative; Animals; Apnea; Body Temperature; Bronchial Provocation Tests; Bronchoconstriction; Bronchoconstrictor Agents; Calibration; Exhalation; Functional Residual Capacity; Humidity; Injections, Intraperitoneal; Ketamine; Lung; Lung Volume Measurements; Methacholine Chloride; Mice; Mice, Inbred BALB C; Models, Biological; Pressure; Radiography; Reproducibility of Results; Respiratory Mechanics; Tidal Volume; Time Factors; X-Rays | 2008 |
Prenatal nicotinic exposure augments cardiorespiratory responses to activation of bronchopulmonary C-fibers.
Topics: Animals; Apnea; Brain-Derived Neurotrophic Factor; Bronchoalveolar Lavage Fluid; Capsaicin; Diaphragm; Electromyography; Female; Ganglia; Humans; Hypoxia; Infant, Newborn; Male; Methacholine Chloride; Nerve Fibers, Unmyelinated; Nerve Growth Factor; Nicotine; Nodose Ganglion; Pregnancy; Prenatal Exposure Delayed Effects; Random Allocation; Rats; Rats, Sprague-Dawley; Receptor, trkA; Receptor, trkB; Recombinant Proteins; Sensory System Agents; Smoking; Substance P; Sudden Infant Death; TRPV Cation Channels | 2015 |
Respiratory dysfunction progresses with age in Kcna1-null mice, a model of sudden unexpected death in epilepsy.
Topics: Animals; Apnea; Bronchoconstrictor Agents; Death, Sudden; Disease Models, Animal; Disease Progression; Epilepsy; Epilepsy, Temporal Lobe; Gene Expression; Hyperventilation; Hypoxia; Kv1.1 Potassium Channel; Methacholine Chloride; Mice; Mice, Knockout; Muscle, Smooth; Respiratory Insufficiency; Reverse Transcriptase Polymerase Chain Reaction; Tachypnea; Tidal Volume; Trachea | 2018 |
Progressive cardiorespiratory dysfunction in Kv1.1 knockout mice may provide temporal biomarkers of pending sudden unexpected death in epilepsy (SUDEP): The contribution of orexin.
Topics: Animals; Apnea; Bradycardia; Epilepsy; Heart Rate; Hypothalamic Area, Lateral; Hypoxia; Kv1.1 Potassium Channel; Methacholine Chloride; Mice; Mice, Knockout; Neurons; Orexin Receptor Antagonists; Orexins; Oximetry; Oxygen; Parasympathetic Nervous System; Parasympathomimetics; Respiratory Rate; Seizures; Sudden Unexpected Death in Epilepsy | 2020 |